Effect of hydrofluoric acid etching time on Ni/6H-SiC contacts

Abstract

The effect of hydrofluoric acid （HF） etching time on Ni/6H-SiC ohmic contacts was investigated． The as-deposited Ni/6H-SiC contacts prepared by 6H-SiC substrates which have been subjected to different HF etching time have different I-V characteristics． For SiC substrates etched for less than 12 hours, the contacts were rectifying, and excellent linear curves were observed after high temperature thermal annealing．X_ray diffraction, Auger electronic spectroscopy and low_energy reflection electron energy loss spectroscopy showed that Ni2Si and amorphous C were the main reaction products after annealing.For SiC substrate etched for 24 hours, the as-deposited Ni/6H-SiC contact was ohmic． The carbon-enriched layer （CEL） on the SiC surface plays an important role in the formation of ohmic contact．

Salicylate Induced GABAAR Internalization by Dopamine D1-Like Receptors Involving Protein Kinase C (PKC) in Spiral Ganglion Neurons

“…Research showed that the cochlear response of the D1 knockout mice was enhanced, and the auditory brainstem response (ABR) threshold of all frequencies was increased by 5–20 dB [ 12 ], indicating that D1Rs play an important role in auditory function. Our previous studies have shown that D1R antagonist can weaken the effect of SS on GABA A R expression in SGNs, suggesting that D1Rs can mediate the effect of SS on GABA A Rs by interacting with GABA A Rs [ 11 , 13 ]. In this work, the expression of DRD1 on SGNs was detected by immunofluorescence, which was in agreement with the reports of Maison et al [ 12 ], Wei et al [ 11 ], and Inoue et al [ 24 ].…”

“…D1Rs have been detected on SGNs [ 11 , 12 ]. SS can promote the mRNA expression of D1Rs in SGNs [ 13 ]. Other research has shown that D1Rs can interact with GABA A Rs and reduce the GABA-evoked currents in the middle spinous neurons of the neostriatum [ 14 ].…”

Allergic Rhinitis and Depression: Profile and Proposal

“…(c) It may be defined as “AR with depression” when it is considered as a co-morbidity. Theoretically this condition does exist, and the possibility of co-morbidity of AR and depression has been suggested in studies on genetic susceptibility in twins ( 3 , 15 , 16 ). However, none of the studies retrieved has independently reported this condition, and we assume that many studies might have classified it into the second category.…”

“…In terms of genetic susceptibility, some Chinese authors ( 3 ) suggested that genetic factors, especially maternal inheritance, may play a key role in the association between AR and depression, and agreed on the possibility that the ADCYAP1R1 gene ( 28 ) deserves attention; however, studies on a single gene are limited, and the potential role of epigenetic research warrants further investigations.…”

“…Although AR itself is not a life-threatening disease, it can significantly affect the quality of life and even lead to reduced work efficiency; in a certain number of patients, it is liable to trigger depression, anxiety, and other mental disorders due to the stress incurred by AR itself or other negative events, which to some extent increases the burden on families and society. It is estimated that 20.6–38.7% of AR patients will have depressive symptoms ( 3 , 4 ); the treatments become more difficult and costly in AR patients with depression ( 5 ). Theoretically AR may interact with depression.…”