2003
DOI: 10.1161/01.str.0000097490.88015.3a
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Effect of Treatment of Carotid Artery Stenosis on Blood Pressure

Abstract: Background and Purpose-Carotid intervention by carotid endarterectomy (CEA) or endovascular treatment may cause hemodynamic change. The immediate and long-term effects on blood pressure after these procedures were assessed. Methods-Patients were randomized to CEA (nϭ49) or endovascular treatment (nϭ55) that comprised percutaneous transluminal angioplasty alone (nϭ31), balloon-expandable stent (nϭ13), or self-expandable stent (nϭ11). A baseline 24-hour ambulatory blood pressure recording was made before carotid… Show more

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Cited by 81 publications
(26 citation statements)
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“…Moreover, the fact that patients were asked to hold antihypertensive medications before the procedure and were routinely administered anticholinergic drugs during the procedure, in addition to the relatively short duration of hemodynamic monitoring, can probably reduce the estimated incidence of hemodynamic depression in this cohort. McKevitt et al 24 demonstrated that, on average, hemodynamic perturbations persist for more than 20 hours.…”
Section: Discussionmentioning
confidence: 99%
“…Moreover, the fact that patients were asked to hold antihypertensive medications before the procedure and were routinely administered anticholinergic drugs during the procedure, in addition to the relatively short duration of hemodynamic monitoring, can probably reduce the estimated incidence of hemodynamic depression in this cohort. McKevitt et al 24 demonstrated that, on average, hemodynamic perturbations persist for more than 20 hours.…”
Section: Discussionmentioning
confidence: 99%
“…In some reports, HI has been associated with neurological sequelae. 3,10,11,20 However, HI does not occur after CAS in all patients; there are numerous debates in the literature regarding the incidence of this phenomenon and possible predictors. In addition, there have been efforts to determine the correlation between HI and increased mortality and morbidity in the short and long term in those who have undergone CAS.…”
Section: Discussionmentioning
confidence: 99%
“…First, the higher incidence of embolisms after CAS (Roh et al, 2005) might explain how a hemorrhagic transformation could occur after the resolution of the embolism in the tissue that was damaged; from a pathophysiological point of view, however, this would represent hemorrhagic infarction due to reperfusion rather than CHS. Second, the higher incidence of bradycardia and hypotension after the stimulation of the carotid baroreceptors during CAS (Mendelsohn et al, 1998;McKevitt et al, 2003;Qureshi et al, 1999) can favor cerebral ischemia and CHS after severe rebound hypertension (Abou-Chebl et al, 2007). In an earlier publication , these authors suggested that SPECT findings of hyperperfusion continuing at least three days after revascularization predisposes to CHS.…”
Section: Extracranial Carotid Angioplastymentioning
confidence: 99%