1980
DOI: 10.1620/tjem.132.453
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Effects of a glucoside-hydrolase inhibitor (Bay g 5421) on serum lipids, lipoproteins and bile acids, fecal fat and bacterial flora, and intestinal gas production in hyperlipidemic patients.

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Cited by 26 publications
(16 citation statements)
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“…1). In this regard, our findings confirm the early report by Maruhama et al 31 who found, by the use of a cultural method, that acarbose (Bay-g-5421) treatment significantly increased the number of bifidobacteria and lactobacilli, and decreased the number of enterobacteriaceae, bacteroidaceae and lecithinase (+) clostridium in patients with dyslipidemia. Consistent with this, Xiao et al 32 showed that type 2 diabetic patients treated with acarbose had increased cultivable numbers of bifidobacteria and decreased numbers of enterobacteriaceae, but no effects on the numbers of Enterococcus faecalis, lactobacilli, or bacteroidaceae were observed.…”
Section: Discussionsupporting
confidence: 92%
See 1 more Smart Citation
“…1). In this regard, our findings confirm the early report by Maruhama et al 31 who found, by the use of a cultural method, that acarbose (Bay-g-5421) treatment significantly increased the number of bifidobacteria and lactobacilli, and decreased the number of enterobacteriaceae, bacteroidaceae and lecithinase (+) clostridium in patients with dyslipidemia. Consistent with this, Xiao et al 32 showed that type 2 diabetic patients treated with acarbose had increased cultivable numbers of bifidobacteria and decreased numbers of enterobacteriaceae, but no effects on the numbers of Enterococcus faecalis, lactobacilli, or bacteroidaceae were observed.…”
Section: Discussionsupporting
confidence: 92%
“…). In this regard, our findings confirm the early report by Maruhama et al . who found, by the use of a cultural method, that acarbose (Bay‐g‐5421) treatment significantly increased the number of bifidobacteria and lactobacilli, and decreased the number of enterobacteriaceae, bacteroidaceae and lecithinase (+) clostridium in patients with dyslipidemia.…”
Section: Discussionsupporting
confidence: 91%
“…Pulmonary hydrogen was collected by the end-expiratory method (Metz et al 1976) at 10 min intervals after test meal ingestion and measured by a Yanaco Type G 1800-T gaschromatograph (Yanagimoto Co., Kyoto) with argon as a carrier gas (Maruhama et al 1980). Blood samples for the determination of plasma motilin concentrations were obtained every 10 min for 120 min before and after the ingestion, and collected into tubes containing aprotinin and EDTA and were kept on ice until separation into plasma by centrifugation.…”
Section: Methodsmentioning
confidence: 99%
“…This suggests that acarbose prevents starch processing and absorption and enhances starch-fermenting and butyrate-producing bacteria, at the same time, it inhibits starch use by propionate-producing bacteria [ 58 ]. Acarbose administration in hyperlipidemic or T2D patients was further shown to increase Lactobacillus and Bifidobacterium [ 23 , 24 , 29 ] as well as other SCFA-producing bacteria such as Faecalibacterium and Prevotella [ 24 ]. Moreover, Zhang and colleagues [ 24 ] showed that the increased abundance of Dialister after acarbose treatment is negatively correlated with HbA1c, which indicates a probable role for species belonging to this taxon in the regulation of glucose metabolism.…”
Section: The Effects Of Antidiabetic Treatments On Gut Microbiota mentioning
confidence: 99%
“…Moreover, Zhang and colleagues [ 24 ] showed that the increased abundance of Dialister after acarbose treatment is negatively correlated with HbA1c, which indicates a probable role for species belonging to this taxon in the regulation of glucose metabolism. Finally, acarbose treatment was also associated with a diminution of Enterobacteriaceae, Bacteroidaceae and lecithinase positive Clostridum in human feces [ 23 ].…”
Section: The Effects Of Antidiabetic Treatments On Gut Microbiota mentioning
confidence: 99%