Effects of Alcohol, Hot Drinks, or Smoking on Hand and Foot Heat Loss

Goldman, Rachel; Newman, Russel W.; Wilson, O. B.

doi:10.1111/j.1748-1716.1973.tb05416.x

Cited by 10 publications

(2 citation statements)

References 15 publications

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“…It is possible that the ingestion of the¯uid caused re¯ex thermoregulatory eects (Goldman et al 1973). However, the ingestion of the hot beverage caused no decrease in " sk , so that the thermoregulatory eects of drinking the beverages are likely to be minimal.…”

Section: Discussionmentioning

confidence: 99%

The effect of body temperature on the hunting response of the middle finger skin temperature

Daanen¹,

Linde²,

Romet³

et al. 1997

European Journal of Applied Physiology

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The relationship between body temperature and the hunting response (intermittent supply of warm blood to cold exposed extremities) was quantified for nine subjects by immersing one hand in 8 degree C water while their body was either warm, cool or comfortable. Core and skin temperatures were manipulated by exposing the subjects to different ambient temperatures (30, 22, or 15 degrees C), by adjusting their clothing insulation (moderate, light, or none), and by drinking beverages at different temperatures (43, 37 and 0 degrees C). The middle finger temperature (Tfi) response was recorded, together with ear canal (Tear), rectal (Tre), and mean skin temperature (Tsk). The induced mean Tear changes were -0.34 (0.08) and +0.29 (0.03) degrees C following consumption of the cold and hot beverage, respectively. Tsk ranged from 26.7 to 34.5 degrees C during the tests. In the warm environment after a hot drink, the initial finger temperature (T(fi,base)) was 35.3 (0.4) degrees C, the minimum finger temperature during immersion (T(fi,min)) was 11.3 (0.5) degrees C, and 2.6 (0.4) hunting waves occurred in the 30-min immersion period. In the neutral condition (thermoneutral room and beverage) T(fi,base) was 32.1 (1.0) degrees C, T(fi,min) was 9.6 (0.3) degrees C, and 1.6 (0.2) waves occurred. In the cold environment after a cold drink, these values were 19.3 (0.9) degrees C, 8.7 (0.2) degrees C, and 0.8 (0.2) waves, respectively. A colder body induced a decrease in the magnitude and frequency of the hunting response. The total heat transferred from the hand to the water, as estimated by the area under the middle finger temperature curve, was also dependent upon the induced increase or decrease in Tear and Tsk. We conclude that the characteristics of the hunting temperature response curve of the finger are in part determined by core temperature and Tsk. Both T(fi,min) and the maximal finger temperature during immersion were higher when the core temperature was elevated; Tsk seemed to be an important determinant of the onset time of the cold-induced vasodilation response.

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Section: Discussionmentioning

confidence: 99%

The effect of body temperature on the hunting response of the middle finger skin temperature

Daanen¹,

Linde²,

Romet³

et al. 1997

European Journal of Applied Physiology

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“…Early investigators noted an increase in peripheral vasodilation and suggested an increase in heat loss accompanied acute ad ministration of ethanol [5,12,13,17,36], However, a number of other reports indicates that there is little change in total body heat loss from man following intake of ethanol at either cold [2,9,14,25] or neutral Ta [20].…”

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confidence: 99%

Response to Thermal Stress in the Rat following Acute Administration of Ethanol

Spiers¹,

Threatte²,

Fregly³

1984

Pharmacology

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Several thermoregulatory responses (i.e., colonic temperature, tail-skin temperature, rate of oxygen consumption) were measured in the rat following acute administration of either saline or 3 g ethanol/kg BW i.p. at ambient temperatures (T_a) of 17, 25, and 32 °C. The magnitude of the ethanol-induced, hypothermic response was inversely related to T_a, with the decrease in colonic temperature (T_co) at 120 min postinjection ranging from 0.1 °C at T_a32 °C to 3.2 °C at T_a 17 °C. Depression of rate of oxygen consumption (heat production) was a major factor contributing to ethanol-induced hypothermia, with no observed differences in heat loss as assessed by differences in the responses of tail skin temperature. Thermoregulatory responses were also measured following acute administration of ethanol at T_a 25 °C and immediate exposure to either T_a 32 or 17 °C. The increase in T_co of ethanol-treated rats was delayed compared to controls following exposure to 32 °C. In addition, tail skin temperature and rate of oxygen consumption of treated rats were significantly lower. The delayed rise in T_co is most likely the result of a reduction in rate of oxygen consumption. When administered ethanol at T_a 25 °C and then exposed to T_a 17 °C, the rats exhibited a significant recovery from the metabolic depression that characterized the administration of ethanol during exposure to T_a 17 ° C. This was most likely related to differences in the clearance of ethanol from blood. The results of this study are consistent with the suggestion that the physiologically significant inhibitory effect of acute administration of 3 g ethanol/kg BW on thermoregulatory responses of rats is manifested at the level of heat production rather than heat loss and that the effect is exacerbated by a reduction in T_a.

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Thermoregulation and Alcohol

Spiers¹

1995

Alcohol and Hormones

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Effects of Alcohol, Hot Drinks, or Smoking on Hand and Foot Heat Loss

Cited by 10 publications

References 15 publications

The effect of body temperature on the hunting response of the middle finger skin temperature

The effect of body temperature on the hunting response of the middle finger skin temperature

Response to Thermal Stress in the Rat following Acute Administration of Ethanol

Thermoregulation and Alcohol

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