“…It is also crucial for emotion regulation (Golkar, Lonsdorf et al 2012, Hartling, Metz et al 2021, Nejati, Majdi et al 2021) and reward processing (Rushworth, Noonan et al 2011, Overman, Sarrazin et al 2023), probably through top-down regulation (Drevets and Raichle 1992, Simpson, Öngür et al 2000). An increase in DLPFC activity has been shown to facilitate emotion regulation and improve cognitive control (Chen, Oei et al 2023), and impaired emotional regulation due to DLPFC dysfunction is closely related to depression, in which the complex interaction between cognition and emotion is significantly disrupted (Salehinejad, Ghanavai et al 2017). Previous studies have repeatedly shown a negative correlation between DLPFC and AMY (Davidson and Irwin 1999, Mayberg, Liotti et al 1999, Damasio, Grabowski et al 2000), demonstrating top-down control of emotion through increased DLPFC recruitment (Bishop 2008, Iordan, Dolcos et al 2013) to down-regulate activity in AMY (Loos, Schicktanz et al 2020, de Voogd and Hermans 2022).…”