Electrocardiographic features of a population of athletes aged between 8 and 16 years: a comparison with the young adult athlete

Piseri, Maria; Halász, Géza; Biasini, Vincenzo; Capelli, Bruno; Nardecchia, Silvia; Fallavollita, Luca; Petroni, Renata; Penco, María; Romano, Silvio

doi:10.23736/s0025-7826.19.03472-0

Cited by 2 publications

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“…The association of vasospasm or endoluminal thrombosis may contribute to the hemodynamic signifi cance and clinical symptomatology of these conditions. In fact, abnormal coronary vasoreactivity has been reported after administration of acetylcholine at the level of the myocardial bridge; this explains both the permanent asymptomaticity of the majority of subjects carrying the anomaly and the variability in the time of onset of symptoms, as well as the broad spectrum of manifestations ascribable to the anomaly in published clinical case histories (stress angina, myocardial infarction, unstable angina, episodes of acute pulmonary edema, ventricular tachycardia, sudden death) [1][2][3][4][5].…”

Section: Introductionmentioning

confidence: 99%

Myocardial bridges and competitive sports fitness between past and future

ASSISI¹,

E²,

Grossgasteiger³

et al. 2023

J Cardiovasc Med Cardiol

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The myocardial bridge is an intramural course of a coronary artery in which a more or less long section of a coronary branch, instead of running normally on the epicardial surface of the heart, deepens early in the myocardium, coming to be surrounded by a ring or sleeve of muscle fiber cells that, contracting in systole, can cause an ab-extrinsic “throttling” of the artery. The guidelines for granting eligibility for competitive sports have evolved over time: from 2009, when any case of myocardial bridging indicated exclusion of eligibility, to today, when only significant bridges, i.e. ‘long’ bridges > 1 cm and > 3 mm deep, place a restriction on competitive sports activity.

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