Embolie de liquide amniotique : évolution favorable après embolisation thérapeutique des artères utérines

Dorne, R.; Pommier, Christian; Emery, J.-C.; Dieudonné, F; Bongiovanni, Jean-Pierre

doi:10.1016/s0750-7658(02)00638-x

Cited by 19 publications

(6 citation statements)

References 15 publications

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“…47 A pressure gradient that favors the entry of amniotic fluid from the uterus into the maternal circulation is thought to be present. 48 Support for this hypothesis comes from the work of Talbert et al, 49 who found that significant amounts of 125 I-labeled albumin were transferred from amniotic fluid into the maternal circulation following intraamniotic injection of hypertonic saline. This mechanism probably explains the increased risk of AFE among women with placenta previa, placental abruption, operative deliveries, cervical laceration or uterine rupture, and polyhydramnios.…”

Section: Resultsmentioning

confidence: 99%

“…Increased serum levels of tryptase, a marker of mast cell degranulation, in women with AFE have been reported by some authors 123,124 but not by others. 83,125 Benson et al 57 reported that serum tryptase was negative in all of the 6 women with AFE. However, they found that decreased serum levels of C3 and C4 complement had sensitivities between 88–100% and a specificity of 100% for the diagnosis of AFE.…”

Section: Diagnosismentioning

confidence: 99%

See 1 more Smart Citation

Amniotic fluid embolism: an evidence-based review

Conde-Agudelo

Romero

2009

American Journal of Obstetrics and Gynecology

245

277

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We conducted an evidence-based review of information bout amniotic fluid embolism (AFE). The estimated incidence of AFE is 1:15,200 and 1:53,800 deliveries in North America and Europe, respectively. The case fatality rate and perinatal mortality associated with AFE are 13–30% and 9–44%, respectively. Risk factors associated with an increased risk of AFE include advanced maternal age, placental abnormalities, operative deliveries, eclampsia, polyhydramnios, cervical lacerations, and uterine rupture. The hemodynamic response in AFE is biphasic, with initial pulmonary hypertension and right ventricular failure, followed by left ventricular failure. Promising therapies include selective pulmonary vasodilators and recombinant activated facto Vlla. Important topics for future research are presented.

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Section: Resultsmentioning

confidence: 99%

Section: Diagnosismentioning

confidence: 99%

Amniotic fluid embolism: an evidence-based review

Conde-Agudelo

Romero

2009

American Journal of Obstetrics and Gynecology

245

277

View full text Add to dashboard Cite

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“…A number of reports have described an increase in the tryptase level, suggesting an association with anaphylaxis (38,39). Other investigators found normal levels of tryptase but low levels of complement (7,40). This may suggest a role for complement activation in amniotic fluid embolism.…”

Section: Diagnosismentioning

confidence: 87%

Amniotic fluid embolism

Moore

Baldisseri

2005

Critical Care Medicine

128

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Patients with amniotic fluid embolus are best managed using a multidisciplinary approach. There are no pharmacologic or other therapies that prevent or treat the amniotic fluid embolism syndrome, and supportive care typically involves aggressive treatment of multiple types of shock simultaneously. In this article we discuss the clinical presentation of amniotic fluid embolism syndrome as well as current opinions regarding pathophysiology, diagnosis, and management.

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“…Plasma exchange transfusions (55,56) 5. Uterine artery embolization (53,57) 6. Continuous hemofiltration (56) 7.…”

Section: Prognosismentioning

confidence: 99%