Enamel Hypoplasia (Chronologic Enamel Aplasia) In Relation to Systemic Disease: A Chronologic, Morphologic and Etiologic Classification

Morphological study of amelogenesis in the rat lower incisor after thyro-parathyroidectomy, parathyroidectomy and thyroidectomy

“…The ameloblasts related to the elevations are more curved than those related to the depressions. Since experimental hypoparathyroidism and hypocalcemia have been shown to result in enamel hypoplasia (Sarnat and Schour 1942), the depressions are probably sites of underproduction of the enamel matrix.…”

In the shadow of Moundville: A bioarchaeological view of the transition to agriculture in the central Tombigbee valley of Alabama and Mississippi

“…Lending more evidence into the social relations of health as it impacts children, generalized stress indicators appear (macro-or microscopically) as lines, pits or furrows on the labial surface of developing upper and lower canines and incisors (Goodman et al, 1980;Hillson, 1996;King et al, 2005;Sarnat and Schour, 1941). Though a specific etiology for enamel hypoplasias (EH) has yet to be identified, they are commonly thought to result from a combination of nutritional deficiencies and systemic infections (El-Najjar et al, 1978;Hillson, 1996;Sarnat and Schour, 1941) including weaning during early childhood (Katzenberg et al, 1996).…”

“…Though a specific etiology for enamel hypoplasias (EH) has yet to be identified, they are commonly thought to result from a combination of nutritional deficiencies and systemic infections (El-Najjar et al, 1978;Hillson, 1996;Sarnat and Schour, 1941) including weaning during early childhood (Katzenberg et al, 1996). Enamel defects were relatively common, although highly variable, in the central Tombigbee sites, with prevalence ranging from 20% to 100% (Table 2).…”

An inter-site comparison of enamel hypoplasia in bison: implications for paleoecology and modeling Late Plains Archaic subsistence

“…A correlation between birth or neonatal stress and hypoplasias has been proposed in humans [50,55] and might be applicable to other mammals; for example, in their early study of enamel hypoplasias Sarnat and Schour [50, pp. 67-68] noted that two-thirds of DEH in their sample formed during the weeks (up to 12) just after birth, as a result of ''postnatal adjustment.''…”