Endoscopic visualization-assisted corneal bee sting removal

Abstract

Purpose: Deeply embedded corneal foreign bodies and intrastromal foreign body removal can often be a challenge. The aim of this report was to describe the utility of endoscopy in visualization and removal of an embedded corneal bee stinger. Methods: A 44-year-old male patient developed toxic keratopathy after injury from a bee stinger. On examination, the bee stinger was noted to be deeply embedded in the corneal stroma. A superficial keratectomy was initially attempted; however, the stinger was noted to be intrastromal and protruding into the anterior chamber and could not be removed. An Endoscopy-assisted visualization was used to remove the stinger. Results: The bee stinger was successfully removed and the patient's vision improved to 20/100 from an initial CFCF (counting fingers close to face) at time of presentation. At the end of 3 months follow-up, there was residual corneal edema along with cataractous changes in the lens as a sequelae of the initial bee sting injury. The patient subsequently underwent an endothelial keratoplasty along with phacoemulsification with intraocular lens implantation and the final BCVA improved to 20/40. Conclusion: Endoscopyassisted visualisation of anterior chamber and angle structures can be valuable in removal of retained and deeply embedded corneal or intracameral foreign bodies.

Phenotypic and Transcriptomics Analyses Reveal Underlying Mechanisms in a Mouse Model of Corneal Bee Sting

“…In the present study, 12 h after stromal injection of PBS, corneal edema subsided and the epithelial defects recovered rapidly; however, mice injected with BV still suffered to some extent from corneal epithelial defects and turbidity after 72 h. Moreover, even following treatment with Tobradex ® , the cornea was thicker than that seen in the VE group, indicating incomplete healing [ 29 ]. Unexpectedly, residual corneal scarring and clouding of the anterior lens capsule is found in clinical cases several years after conventional treatment [ 39 ], and the density of endothelium in the affected eye is significantly reduced [ 29 ]. Therefore, it is imperative to investigate the pathogenesis of CBS with a view to exploring novel drug targets and biomarkers.…”