Endothelial Cell Damage after Intracapsular Cataract Extraction and Primary Anterior Chamber Pseudophakos Implantation

Neetens, A; Dierens, Melissa; Delgadillo, R. A.

doi:10.1159/000309309

Cited by 2 publications

(1 citation statement)

References 5 publications

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“…Roper-Hall and Wilson (43) emphasize that the major cause of cell loss in eyes with intraocular lens implants is direct operative trauma. Despite this the corneal endothelium may still undergo changes a considerable time after an episode of trauma and in this regard Neetens et al (36) stress the importance of long term follow-up. Kaufman and Katz (22) attribute increased endothelial cell morbidity in cases of pseudophakia, as opposed to simple aphakia, to cellular membrane damage induced by intraocular lens/corneal touch.…”

Section: Discussionmentioning

confidence: 99%

Long term observations on an anterior chamber Ridley intraocular lens

Jackson¹,

Archer²,

Chakravarthy³

et al. 1986

Int Ophthalmol

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In 1965 the patient, aged 6, sustained a perforating eye injury which was repaired and a traumatic cataract was aspirated within five weeks. Five years later a Ridley Mk 2 A/C intraocular lens was inserted. Several episodes of blunt trauma occurred over a three year period following this procedure. This paper reports the clinical, corneal pachometric and specular microscopic findings of both traumatized and normal fellow eyes 18 years after the initial incident. The corneal endothelial mosaic of the traumatized right eye was very irregular in the vicinity of the initial site of perforation. These marked variations in cell size and shape were less apparent at peripheral corneal areas. The estimated cell loss to the traumatized eye was in the region of 74% although in spite of this corneal function was maintained. The effects of trauma on the corneal endothelium are discussed and a brief review of the literature presented.

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Section: Discussionmentioning

confidence: 99%

Long term observations on an anterior chamber Ridley intraocular lens

Jackson¹,

Archer²,

Chakravarthy³

et al. 1986

Int Ophthalmol

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Complications of anterior chamber lens implants and their effects on the endothelium

Walters¹,

McGill²,

Batterbury³

et al. 1989

Eye

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A five-year follow-up study of 186 eyes which were subject to intracapsular cataract extraction and rigid anterior chamber lens implantation is reported. Ninety-two per cent of cases achieved a visual acuity of 6/12 or better. Serial corneal endothelial cell counts were performed. In 37 pairs of eyes the median difference in operative endothelial cell loss between the operated and un-operated eyes was -7.3% (95% Confidence Interval from -8.7 to -6.1%). Iris tuck occurred in 22.0% and late 'iris ovalling' developed in a further 15.6% of eyes. The median operative endothelial cell loss for those eyes with iris tuck was -9.7% and this was significantly higher than that of eyes with no long-term complications (median loss = -7.3%). The median operative endothelial cell loss for those eyes which developed late ovalling was -8.0% and this was not significantly greater than that of the eyes with no long-term complications (p = 0.745). There was a progressive median endothelial cell loss for all operated eyes and this progressive cell loss was significantly greater for those eyes with iris tuck when compared with those with no complications (p = 0.045).

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Endothelial Cell Damage after Intracapsular Cataract Extraction and Primary Anterior Chamber Pseudophakos Implantation

Abstract: There is a gradual reduction in endothelial cell count with increasing age, but there is no significant difference in endothelial cell loss between aphakic eyes and pseudo-phakic eyes, the percentage being 33.14 and 26.28%, respectively.

Cited by 2 publications

References 5 publications

Long term observations on an anterior chamber Ridley intraocular lens

Long term observations on an anterior chamber Ridley intraocular lens

Complications of anterior chamber lens implants and their effects on the endothelium

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