Enlargement of internal carotid artery aneurysm presenting with severe visual sequela: A case report and anatomy review

Roberts, Amanda Mendez; Grimes, Amy L.

doi:10.1016/j.optm.2008.05.009

Cited by 11 publications

(7 citation statements)

References 8 publications

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“…However, as Kim and Park [ 38 ] suggest, clinicians should consider ICA aneurysms when patients present isolated AN palsy. Mendez Roberts and Grimes [ 39 ] emphasize, in turn, that the proximity of cranial nerves II to VI to the course of the ICA makes these nerves susceptible to damage from an ICA aneurysms. According to Elder et al [ 40 ], the etiologies of cavernous AN segment palsy include: cavernous part of the ICA aneurysm, neoplastic infiltration, idiopathic inflammation (Tolosa-Hunt syndrome), infection, and cavernous-carotid fistula.…”

Section: Discussionmentioning

confidence: 99%

Gross and Micro-Anatomical Study of the Cavernous Segment of the Abducens Nerve and Its Relationships to Internal Carotid Plexus: Application to Skull Base Surgery

et al. 2021

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The present study aims to provide detailed observations on the cavernous segment of the abducens nerve (AN), emphasizing anatomical variations and the relationships between the nerve and the internal carotid plexus. A total of 60 sides underwent gross-anatomical study. Five specimens of the AN were stained using Sihler’s method. An additional five specimens were subjected to histological examination. Four types of AN course were observed: a single nerve along its entire course, duplication of the nerve, division into separate rootlets at the point of contact with the cavernous part of the internal carotid artery (ICA), and early-branching before entering the orbit. Due to the relationships between the ICA and internal carotid plexus, the cavernous segment of the AN can be subdivided into a carotid portion located at the point of contact with the posterior vertical segment of the cavernous ICA and a prefissural portion. The carotid portion of the cavernous AN segment is a place of angulation, where the nerve always directly adheres to the ICA. The prefissural portion of the AN, in turn, is the primary site of fiber exchange between the internal carotid plexus and either the AN or the lateral wall of the cavernous sinus.

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Section: Discussionmentioning

confidence: 99%

Gross and Micro-Anatomical Study of the Cavernous Segment of the Abducens Nerve and Its Relationships to Internal Carotid Plexus: Application to Skull Base Surgery

et al. 2021

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“…12,15,18 However, without any treatment, no change in visual impairment or progressive visual deterioration had previously been reported. 4,19 We recently reported on posterior communicating artery aneurysm-related oculomotor nerve palsy, including a systematic review, and found significantly higher resolution of oculomotor nerve palsy after surgical treatment. 11 Due to discrepant literature data, in this study we analyzed the outcome of visual dysfunction caused by intracranial aneurysms after surgical and endovascular treatment at our institution.…”

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confidence: 99%

Influence of surgical or endovascular treatment on visual symptoms caused by intracranial aneurysms: single-center series and systematic review

Schuss¹,

Güresir²,

Berkefeld

et al. 2011

JNS

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“…Other tumours include craniopharyngiomas and pituitary adenomas. Compression of the optic nerve by a growing internal carotid aneurysm can cause visual loss and proptosis31 32 (figure 3). Other less common benign masses have been reported to present with loss of vision due to compression of the optic nerve, such as myofibromas of the orbit, osteopetrosis, pyknodysostosis, Rathke's cleft cyst, clinoid mucocele, fibrous dysplasia of the skull, and sphenoid sinus plasmacytoma 33–37.…”

Section: Aetiologiesmentioning

confidence: 99%

Acute neurological visual loss in young adults: causes, diagnosis and management

Ayoubi

Hamam²

2015

Postgraduate Medical Journal

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Visual loss in the young adult can be caused by demyelinating diseases, inflammatory and autoimmune processes, infections, ischaemic events, and compressive lesions of the optic nerve. Diagnosis of the aetiologies of visual loss is reached by combining data from radiological studies, electrophysiological tests, and blood and cerebrospinal fluid analysis. Treatment is primarily aimed at decreasing the insult on the optic nerve and eventually controlling the primary disorder. The literature discusses separately the different aetiologies of visual loss. We present a review of the clinical characteristics of visual loss in the young adult, the different diagnostic measures, and the latest therapeutic strategies. The aim of this work is to summarise this entity in a practical way to guide clinicians in the diagnosis and management of this disorder.

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Enlargement of internal carotid artery aneurysm presenting with severe visual sequela: A case report and anatomy review

Cited by 11 publications

References 8 publications

Gross and Micro-Anatomical Study of the Cavernous Segment of the Abducens Nerve and Its Relationships to Internal Carotid Plexus: Application to Skull Base Surgery

Gross and Micro-Anatomical Study of the Cavernous Segment of the Abducens Nerve and Its Relationships to Internal Carotid Plexus: Application to Skull Base Surgery

Influence of surgical or endovascular treatment on visual symptoms caused by intracranial aneurysms: single-center series and systematic review

Acute neurological visual loss in young adults: causes, diagnosis and management

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