Enzymic determination of urinary oxalate, with EDTA added to improve recovery.

Cannon, G D; Eaton, R H; Glen, A.I.M.; Moncur, J. G.

doi:10.1093/clinchem/29.10.1855

Cited by 10 publications

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“…They added 20 mL of 6 mol/L HCl to the container after collecting the first sample. Urinary oxalate was measured enzymatically using an oxalic acid kit (Boehringer Mannheim, Germany) as described previously [8]. The relationships were then assessed between urinary oxalate excretion and dietary intake of animal fat, various fatty acids and calcium.…”

Section: Methodsmentioning

confidence: 99%

Association of dietary fatty acids with urinary oxalate excretion in calcium oxalate stone‐formers in their fourth decade

et al. 2002

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Objective To examine the influence of the dietary intake of fatty acid on urinary oxalate excretion in calcium oxalate stone-formers in their fourth decade, as previous reports show that animal fat intake is associated with urinary oxalate excretion. Patients and methods The dietary intake of 58 idiopathic stone-formers in their fourth decade was recorded using the dietary-record method. The patients collected 24-h urine samples at home and their urinary oxalate excretion was measured in a clinical biochemistry laboratory. The results were used to determine the relationship between the dietary intake of fatty acids and urinary oxalate excretion. Associations between urinary oxalate excretion and dietary contents of animal fat, animal protein and various fatty acids were assessed using Spearman's correlation coefficient and multiple regression. Results The dietary content of arachidonic acid was positively correlated with urinary oxalate excretion, as assessed by univariate and multivariate analysis. Conclusion The association between arachidonic acid and oxalate excretion suggests that arachidonic acid increases the intestinal absorption of oxalate and increases the clearance of oxalate in the kidneys.

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Section: Methodsmentioning

confidence: 99%

Association of dietary fatty acids with urinary oxalate excretion in calcium oxalate stone‐formers in their fourth decade

et al. 2002

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“…Urinary citrate was measured enzymatically using the Boehringer Mannheim citric acid kit (Warty et al, 1984). Urinary oxalate was measured enzymatically using the Boehringer Mannheim oxalic acid kit (Cannon et al, 1975). Calcium: creatinine ratios were performed according to the method of Pak et al (1975).…”

Section: Methodsmentioning

confidence: 99%

Hyperoxaluria in Patients with Recurrent Calcium Oxalate Calculi: Dietary and Other Risk Factors

Laminski

Meyers

Kruger

et al. 1991

British Journal of Urology

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The presence of mild hyperoxaluria in recurrent calcium oxalate stone formers is controversial. The aim of this study was to identify recurrent stone formers with mild hyperoxaluria and to classify them further by assessing their response to a low oxalate diet. In addition, the prevalence of other risk factors for stone formation in this group of patients was investigated. A total of 207 consecutive patients with recurrent renal calculi were screened and 40 (19%) were found to have mild hyperoxaluria. Of these, 18 (45%) responded to dietary oxalate restriction by normalising their urinary oxalate. The remaining 22 patients were classified as having idiopathic hyperoxaluria and were subdivided into those in whom urinary oxalate excretion was consistently elevated in all specimens measured and those in whom the elevation was intermittent in nature. Dietary oxalate restriction had a partially beneficial effect in lowering oxalate excretion in the patients with persistent hyperoxaluria. No difference in urinary oxalate excretion was found after dietary restriction in the patients with intermittent hyperoxaluria. Other risk factors, including dietary, absorptive and renal hypercalciuria and hypocitraturia, were documented, the prevalence of which (65%) was not significantly different from that (62.5%) found in 40 age- and sex-matched calcium stone formers without hyperoxaluria. The prevalence of hyperuricosuria was significantly greater in patients with hyperoxaluria when compared with stone controls. Further studies are required to elucidate the underlying mechanisms of hyperoxaluria in recurrent stone formers.

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