Epidermoid Tumour of the Posterior Fossa Causing Multiple Facial Pain a Case Report

Levyman, Celio; Filho, AméRico Dos Santos P. Dágua; Volpato, Margarete Mota; Settanni, Flávio Aurélio Parente; Lima, Wanderley Cerqueira de

doi:10.1046/j.1468-2982.1991.1101033.x

Cited by 20 publications

(14 citation statements)

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“…Secondary, or symptomatic cluster headache has been described from pituitary adenoma [1, 2], meningiomas of the sphenoid wing [3], upper cervical area [4], and undersurface of the tentorium cerebelli [5] (where inflammatory myofibroblastic tumor has also caused cluster headache) [6], aneurysms of the anterior cerebral [1], posterior cerebral [7], vertebral [8], and carotid arteries [1], dissections of the vertebral [9] and internal carotid arteries [10, 11], arteriovenous malformations in the middle cerebral artery [12] as well as occipital [13], frontal, temporal, and parietotemporal lobes [14–16], Wallenberg’s syndrome [17], pituitary orbitosphenoidal aspergillosis [18], head and neck injury [19], ipsilateral enucleation [20], calcified lesions close to the third ventricle [21], cavernous sinus granulomatous tissue [22], and post carotid endarterectomy [23, 24]. …”

Section: Discussionmentioning

confidence: 99%

Cluster headache associated with moyamoya

2008

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A 34-year-old man with right-sided cluster headache presented with a stroke from right-sided moyamoya. Following surgery on the right, both moyamoya and cluster headache remitted, but eighteen months later a cluster attack and symptoms of cerebral ischemia from moyamoya recurred on the left. Again, following surgery on the left, both moyamoya symptoms and cluster attacks disappeared. Cluster headache secondary to moyamoya has not previously been described.Electronic supplementary materialThe online version of this article (doi:10.1007/s10194-008-0081-3) contains supplementary material, which is available to authorized users.

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Section: Discussionmentioning

confidence: 99%

Cluster headache associated with moyamoya

2008

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“…symptomatic clusterlike headache has been reported in a variety of pathological conditions with varying locations. [2][3][4][5][6][7][8][9][10][11][12][13][14][15][16] Recently, pathophysiological interest has been focused on basal midline structures and in particular, the cavernous sinus. [17][18][19][20][21][22][23] We report a case of clusterlike headache associated with an intrasellar pituitary adenoma causing acromegaly.…”

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confidence: 99%

Clusterlike Headache in a Patient With a Pituitary Adenoma. With a Review of Literature

1996

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A man had left-sided atypical clusterlike headache for II years before he developed symptoms and signs consistent with acromegaly. Preoperative evaluation revealed raised levels of somatomedin C and growth hormone. An MR indicated a left-sided intrasellar mass measuring 8 x 7.5 x 10 mm. He underwent surgery and microscopy confirmed the diagnosis of a benign hypophyseal adenoma. Postoperatively, the acromegalic features regressed, and for the last 4 years the patient has been completely free from headache attacks. On pharmacological testing of the pupillary response to 19 and 5% phenylephrine and 2% tyramine solutions, there was no convincing evidence of persistent sympathetic dysfunction on the earlier symptomatic side.Key words: acromegaly, clusterlike headache, phenylephrine pituitary adenoma, pupil, tyramine Abbreviations: GH growth hormone (Headache 1996;36:184-188) The pathophysiology of cluster headache remains unclear. In most instances, every extensive neurological and neuroradiological examinations will be normal. 1 However. symptomatic clusterlike headache has been reported in a variety of pathological conditions with varying locations. [2][3][4][5][6][7][8][9][10][11][12][13][14][15][16] Recently, pathophysiological interest has been focused on basal midline structures and in particular, the cavernous sinus. [17][18][19][20][21][22][23] We report a case of clusterlike headache associated with an intrasellar pituitary adenoma causing acromegaly. The relevance of the tumor location and its possible implications for the symptomatology, particularly with respect to sympathetic dysfunction, are discussed. CASE HISTORYA 37-year-old man had suffered from left-sided headache attacks for 4 years before he sough medical advice at the Department of Neurology Malmö University Hospital in 1985. His previous history was unremarkable. The headache was strictly left-sided and located behind the eye; radiating towards the left ear, the occiput, and the neck. Because the pain also involved the teeth in the left upper jaw, he had undergone dental treatments, including correction of bite, all of which had been in vain.The attacks occurred with varying intervals of weeks or months. The pain was excruciating, with a stinging and pulsating character. It always started at about the same time in the afternoon and lasted for up to 7 or 8 hours. The patient could only get some relief by a high intake of phenacetin and acetylsalicylic acid. However, by pouring hot water over the neck or by massaging the occipital region on the left side, he could also obtain considerable relief. The pain never sub sided completely before he went to bed. Initially, when the headache attacks started, he thought that attacks could be provoked by intense sunlight, shimmering waves, but also by a cold wind blowing in the face. Whenever he suffered an attack, his left eye was reddened with markedly increased lacrimation. The left nostril was blocked and there was rhinorrhea. During an attack, he did not experience nausea, nor did he ever vomit. Som...

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“…To our knowledge, only one case of GPN had been reported among those of multiple facial pains caused by an epidermoid tumour in the posterior fossa. 13 This is the first case where epidermoid tumour led to the development of GPN alone.…”

Section: Discussionmentioning

confidence: 85%