“…That transient amnesia episodes could be related to a nonepileptic cause has also been advocated. Finally, and perhaps most importantly, the clinical relevance of delineating a new epileptic entity within an already dense epilepsy landscape has been questioned [10]. In this article, we will review the main clinical and electrophysiological characteristics of TEA, discuss its neuroanatomical substrate and putative pathophysiological mechanisms, with the overall aim to defend the idea that TEA deserves individualization as a distinct epilepsy syndrome.…”