Epstein–Barr Virus and Burkitt’s Lymphoma

Banerjee, Shuvomoy; Jha, Hem Chandra; Cai, Qiliang; Robertson, Erle S.

doi:10.1007/978-1-4614-4313-1_10

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Burkitt's Lymphoma1

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2013

2016

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Cited by 4 publications

(2 citation statements)

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“…The critical event in the BL oncogenesis is a chromosomal translocation that brings the c-myc oncogene on chromosome 8 under the transcriptional control of one of the immunoglobulin genes on chromosome 14, 2 or 22. Together with malaria or HIV infection, EBV may favor the translocation by inducing B-cell hyperproliferation (reviewed in(93)).…”

mentioning

confidence: 99%

The Gly–Ala repeat modulates the interaction of Epstein–Barr virus nuclear antigen-1 with cellular chromatin

Coppotelli¹,

Mughal²,

Masucci³

2013

Biochemical and Biophysical Research Communications

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mentioning

confidence: 99%

The Gly–Ala repeat modulates the interaction of Epstein–Barr virus nuclear antigen-1 with cellular chromatin

Coppotelli¹,

Mughal²,

Masucci³

2013

Biochemical and Biophysical Research Communications

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“…This then increases the occurrence of one or another of three specific chromosomal translocations, each of which can activate the c‐myc oncogene whose deregulation causes malignant change in the affected cell and the emergence of a tumour clone 17,18 . But c‐myc expression alone does not seem to be sufficient for malignancy 16,19,20 and the co‐operation of some additional gene appears to be necessary.…”

Section: Burkitt's Lymphomamentioning

confidence: 99%