2015
DOI: 10.1038/ncomms9802
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Erratum: STAT3 regulated ARF expression suppresses prostate cancer metastasis

Abstract: Nature Communications 6: Article number:7736 (2015); Published: 22 July 2015; Updated: 26 October 2015 The affiliation details for Jan Pencik are incorrect in this Article. The correct affiliation details for this author are given below: Ludwig Boltzmann Institute for Cancer Research, Waehringerstrasse 13A, 1090 Vienna, Austria.

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Cited by 9 publications
(6 citation statements)
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“…Direct or indirect targeting the STAT3 signaling pathway has been explored in order to counteract cancer [139]. It is thought that hyperactive STAT3 is oncogenic in PCa [140] and has been observed in around 50% of PCa [141]. Moreover, high STAT3 activation is associated with increased PCa malignancy with high Gleason score [141].…”
Section: In Vitro Studiesmentioning
confidence: 99%
“…Direct or indirect targeting the STAT3 signaling pathway has been explored in order to counteract cancer [139]. It is thought that hyperactive STAT3 is oncogenic in PCa [140] and has been observed in around 50% of PCa [141]. Moreover, high STAT3 activation is associated with increased PCa malignancy with high Gleason score [141].…”
Section: In Vitro Studiesmentioning
confidence: 99%
“…Ferguson's study suggests that STAT3 controls NF-κB-induced IL-8 expression by sequestering NF-κB within the cytoplasm, thereby inhibiting IL-8-mediated myeloid tumor in ltration and tumor vascularization and enhance tumor progression [40] . The study results of of Pencik et al also suggest the loss of STAT3 signaling disrupts the ARF-Mdm2-p53 tumor suppressor axis bypassing senescence and occurs with high frequency in PCa metastases [41] . Our results showed that STAT3 expression is signi cantly associated with improved PFS and OS, and is proven to be an independent prognostic factor only for TNBC with low ATM expression.…”
Section: Discussionmentioning
confidence: 88%
“…The STAT3 protein is widely expressed in different types of human tissues and cells, and is involved in cell proliferation, differentiation, apoptosis, and a variety of physiological functions 17 . It is also associated with the physiological and pathological functions of inflammation, tumor, and immune response 18 19 20 . To test whether the self-induction effect of hTFF3 was affected by the STAT3 binding site at −1417 bp to −1409 bp, we first performed ChIP on the hTFF3 promoter (pGL3−1826), which is the best way to study the binding activity in vivo between transcription factors and promoters.…”
Section: Discussionmentioning
confidence: 99%