2016
DOI: 10.1016/j.mvr.2016.04.011
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Erythrocyte deformability — A partner of the inflammatory response

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Cited by 51 publications
(30 citation statements)
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“…Thus, emodin-induced cell membrane scrambling was in part, but not exclusively triggered by entry of extracellular Ca 2+ . Erythrocyte cell membrane scrambling is further stimulated by oxidative stress [118]. Reactive oxygen species (ROS) was thus quantified utilizing 2′,7′-dichlorodihydrofluorescein diacetate (DCFDA).…”
Section: Resultsmentioning
confidence: 99%
“…Thus, emodin-induced cell membrane scrambling was in part, but not exclusively triggered by entry of extracellular Ca 2+ . Erythrocyte cell membrane scrambling is further stimulated by oxidative stress [118]. Reactive oxygen species (ROS) was thus quantified utilizing 2′,7′-dichlorodihydrofluorescein diacetate (DCFDA).…”
Section: Resultsmentioning
confidence: 99%
“…The abnormality of WBV can increase the resistance of blood fluidity and slow down the blood flow velocity, thus leading to the cardiovascular risk. Hct is the most important factor for WBV [2932]. In Table 2, compared with model group, BSP-1b, BSP-2 and BSP-3 could significantly decrease WBV at all shear rates (0.001 <  p  < 0.05, p  < 0.001), as well as BSP-1b, BSP-2 and BSP-3 could significantly decrease Hct ( p  < 0.001).…”
Section: Resultsmentioning
confidence: 99%
“…Adherance of phosphatidylserine exposing erythrocytes to the vascular wall [92,93] may further stimulate blood clotting and trigger thrombosis [94][95][96], thus compromising microcirculation [76,94,[97][98][99][100].…”
Section: Discussionmentioning
confidence: 99%