“…Whiplash neck injury is characterized by severe stretch of the soft tissues of the neck anterior to the cervical spine during the hyperextension phase of the whiplash trauma [Davis 2000;Uhrenholt 2011], and is routinely classified according to the Quebec Task Force Clinical Classification, staging patients in 5 WAD grades based on the severity of signs and symptoms following whiplash (neck pain, musculoskeletal deficits, neurological deficits, and bone fractures/dislocations) [Spitzer 1995]. Although the majority of whiplash injuries demonstrate little or no evidence of severe tissue damage with diagnostic imaging, the posttraumatic tissue edema, small-scale hematoma, and microtrauma of target structures may reflect in severe dysfunction of vital neck organs, such as the esophagus, trachea, carotid artery, jugular vein and neural system [Brademann 1998;Davis 2000;Omar 2007;Uhrenholt 2011]. In our otherwise healthy patient, with no prior history of fainting spells, the alteration, or loss of consciousness upon swallowing solid foods first appeared two weeks after the whiplash injury, which strongly suggested that stretchinduced injury of mechanoreceptors and vagal fibers acquired during car accident related neck trauma might be the fundamental pathophysiological mechanism in this clinical case.…”