were stratified by gender, age, and angina (n=2502) vs. nonangina (n=46,358) (angina, 43.94-43.96 vs. 34.42-34.50; nonangina, 32.43-32.43 vs. 17.25-17.25) and 100-129 mg/dL (angina, 32.12-32.14 vs. 35.10-35.18; nonangina, 53.86-53.86 vs. 32.44-32.44). More women than men had LDL-C ≥130 mg/dL (angina, 27.68-27.72 vs. 23.91-23.93; nonangina, 38.70-38.70 vs. 35.38-35.39). Women were less likely than men to receive statins (angina, 69.95-69.99 vs. 82.11-82.13; nonangina, 59.80-59.80 vs. 63.72-63.72), any antilipidemic medication at all (angina, 25.93-25.97 vs. 13.48-13.48; nonangina, 36.73-36.73 vs. 30.73-30.73), or to have current cholesterol measurements (angina, 56.82-56.88 vs. 34.54-34.56; nonangina, 45.77-45.77 vs. 39.75-39.75 C oronary heart disease (CHD) is the single leading cause of death in both men and women in the United States, accounting for more than one out of every five deaths in 2001.1 Hypertension is one of the most significant risk factors for development of CHD, affecting over half of all people who have suffered a first heart attack, and preceding development of congestive heart failure in 91% of cases. 2,3 While the health problems presented by CHD and hypertension have long been recognized in men and older patients, the magnitude of the burden of these diseases in women and younger patients has been largely overlooked until recent decades.4-6 While women do have some health advantages over men regarding the development of heart disease, these advantages are demonstrably not universal-and once CHD is manifest, women lose prognostic superiority over men. 7,8 Elevated low-density lipoprotein cholesterol (LDL-C) levels (dyslipidemia) are well documented as a major contributor to the development of atherosclerosis and subsequent coronary disease.
9Hypertensive patients with dyslipidemia are at increased risk for formation of atherosclerotic