Etiology, diagnosis and treatment of infectious esophagitis

Rosołowski, Mariusz; Kierzkiewicz, Maciej

doi:10.5114/pg.2013.39914

Cited by 46 publications

(77 citation statements)

References 26 publications

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“…Co-infection is common [17]. Esophageal candidiasis and herpes esophagitis due to the immunosuppressive effects of swallowed fluticasone is well reported in the literature [18][19][20].…”

Section: Infective Esophagitismentioning

confidence: 96%

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Infective Esophagitis: Inhaled Steroids Treatment for Eosinophilic Esophagitis not Always Safe

Zala¹,

Wauters²,

Vanuytsel³

et al. 2017

GHOA

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“…Co-infection is common [17]. Esophageal candidiasis and herpes esophagitis due to the immunosuppressive effects of swallowed fluticasone is well reported in the literature [18][19][20].…”

Section: Infective Esophagitismentioning

confidence: 96%

“…Endoscopy in CMV esophagitis shows well-demarcated large linear and deep ulcer with or without pseudo membrane formation. Intra-nuclear or intra-Cytoplasmic viral inclusions, typical owls eye Cowdry inclusions are seen on histology [17,21].…”

Section: Infective Esophagitismentioning

confidence: 99%

Infective Esophagitis: Inhaled Steroids Treatment for Eosinophilic Esophagitis not Always Safe

Zala¹,

Wauters²,

Vanuytsel³

et al. 2017

GHOA

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“…Management should be with systemic antifungals (eg fluconazole 50-100 mg daily for 1-2 weeks). 17 Endoscopic appearances of circumscribed punched-out oesophageal ulcers with raised edges are characteristic of HSV oesophagitis, confirmed by biopsies showing multinucleate giant cells, with ground-glass nuclei and eosinophilic inclusions. Aciclovir is the treatment of choice.…”

Section: Oesophageal Infectionmentioning

confidence: 99%

“…17 The commonest agent is Candida albicans. Two other important organisms are cytomegalovirus (CMV) and herpes simplex virus (HSV).…”

Section: Oesophageal Infectionmentioning

confidence: 99%

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Acute oesophageal symptoms

Hossain

Caestecker

2015

Clinical Medicine

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Acute oesophageal symptomsAuthors: A specialist registrar in gastroenterology, University Hospitals of Leicester, Leicester, UK; B consultant gastroenterologist, University Hospitals of Leicester, Leicester, UK and peptic stricture are the commonest abnormalities [2][3][4] (Table 1), followed by eosinophilic oesophagitis (EOE), which accounts for over 50% of food impactions in some published case series 2 (Fig 1). Less common causes are achalasia (Fig 2), reflux oesophagitis, extrinsic oesophageal compression and oesophageal cancer.Food bolus impactions are acute events immediately recognised by sufferers. Impactions at cricoid level may be Acute oesophageal symptoms include acute dysphagia or food bolus impaction (most commonly due to strictures, Schatzki ring and eosinophilic oesophagitis), acute chest pain with odynophagia due to oesophageal infections, motility disorders and acute oesophageal rupture (of which oesophageal intramural haematoma is a subtype). Acute full thickness oesophageal rupture carries a high mortality if not recognised early; the clinical features and conditions with which this may be confused are presented and discussed. Acute oesophageal symptoms: definitions and causesThis review covers presentation and management of acute dysphagia, odynophagia and oesophageal chest pain.Dysphagia means 'difficulty in swallowing', distinguished from odynophagia ('pain on swallowing') by a sensation of bolus arrest/ delay, localised anywhere between cricoid and xiphisternum. The sensation of obstruction is associated with swallowing, differentiated from globus -a 'blockage' or 'lump' between cricoid and sternal notch independent of swallowing, typically persisting for hours 1 -and early satiety. The latter is often described as food 'lying' in the epigastrium or the retrosternal area but differing from dysphagia, which arises during swallowing, whereas the fullness of early satiety builds during a meal, eventually inhibiting intake. Food bolus obstruction is sudden-onset total dysphagia, discussed in more detail below.Oesophageal pain resembles other intra-thoracic/upper abdominal visceral pain (eg heart, aorta, gallbladder or lungs), not surprisingly since sensory pathways from these organs and chest wall enter the thoracic spinal cord at similar levels. Thus, oesophageal pain may occur in the chest, epigastrium, back, jaws or arm(s), and is more likely when dysphagia or odynophagia are present. Common causes of acute oesophageal symptoms are listed in Box 1. Food bolus impactionFood bolus impaction is commonest in younger adults. In total, 88-97% of all patients with food bolus impaction have underlying oesophageal abnormalities.

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Esophagitis associated with multimodality management of pediatric Ewing sarcoma of thorax

Agarwal¹,

Logie

Morris

et al. 2018

Pediatric Blood & Cancer

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Approximately one-third of patients with Ewing sarcoma of the thoracic region will develop acute esophagitis. An esophageal D5cm dose < 15 Gy and maximal esophageal dose < 47 Gy may keep the rate of acute esophagitis under 5%. However, the association with neutropenia and consistent response to antifungal therapy suggest chemotherapy-associated toxicity and an infectious component as part of the process.

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Etiology, diagnosis and treatment of infectious esophagitis

Cited by 46 publications

References 26 publications

Infective Esophagitis: Inhaled Steroids Treatment for Eosinophilic Esophagitis not Always Safe

Infective Esophagitis: Inhaled Steroids Treatment for Eosinophilic Esophagitis not Always Safe

Acute oesophageal symptoms

Esophagitis associated with multimodality management of pediatric Ewing sarcoma of thorax

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