Evaluation of antioxidant and insecticidal effects of different ginger extratcs

The effect of Some hepatic worms on adult sheep liver

“…Those results are consistent with what mentioned [19], There is an appearance of bleeding on the surface of liver with precise hemorrhagic foci on the parietal capsoler which is considered as a place of entrance of the previously puerile parasite [11]. As Compatible with previous research [14] evidence that the liver is rigid and rigid with enlargement and thickness in the bile ducts and their inflammation in addition to the existence of adult worms in the pipeline, and it may be due to interaction of immune cells and lymphocyte infiltration and macrophages, which It works to replace the fibrous tissue of the dead areas during later stages of infection as shown in figure (1), due to gradual irritation secration metarial from parasite bile ducts, such as excessive bile ductitis, thickening of duct wall and their emergence clearly with an expansion of the bile cavity and containment of these ducts with liquid viscous materials from Mocha Yeh with yellow material and wall thickening of the yellow bag and large size. [21] Hepatic nodules and multiple foci have also been observed in livers of sheep, which may be caused by an abnormal immune response of host against dead larvae or worm products retained in the liver.…”

“…In the present study the granular lesion indicates the limited focal grouping of epithelial cells and singlecore cells, which are coated with fibrous connective tissue of the capsule This lesion is an immune response of the host against the eggs of worms or it may be dead larvae trapped in the liver as indicated by [23]. The places of movement and wandering of non-adult worms within the liver board were distinguished as they represent the acute phase These place were characterized by their congestion with the presence of diffuse hemorrhagic spots that lead to damage and breakdown of its tissues and thus to reduce anaerobic oxygen, which allowed closterid to multiply by divorce its toxins and induce cell necrosis [14]. Histological test also showed up the existence of many cells of citrus eosinophils with a small number of lymphocytes with associated hemorrhages and edema in hepatic worms.…”

“…The severity of infection depends on several factors, including the immune status of the animal, the nature of the liver tissue, the type of animal, the age and nature of animal feeding, and the number of infectious stages ingested by the final host [13]. The researchers classified the disease, depending on the length of exposure and the clinical symptoms of the disease in to three phases (acute, subacute and chronic) [14,15]. It must be taken into consideration that the acute histoplastic changes showed the presence of fibrous inflammation around the capsule and damage and destruction of hepatic cells with hemorrhagic pathways.…”

Preparation of Foot and Mouth Disease trivalent vaccine type A, O, SAT2 and determination of the Guinea pig protective dose 50 (GPPD50)

“…protective antibody titer (1.5) extended from 2 to 32 Testing of Guinea pigs serum samples, to confirm WPV. From these results, the antibody titer against the the results of GPPD using SNT, showed antibody three serotypes were found to be higher than the 50 recommended titer (1.5) which extended for 32 WPV titers of 1.56, 1.68 and 1.68 log /ml against FMDV and antibody titer of the Guinea pigs of the prepared (1.5 log for SNT and 1.9 log for ELISA) for both of 10 10 th trivalent vaccine with 146S (2.2 µg/serotype/dose) type A and O as estimated by SNT and ELISA on the 4 these results come in agreement with previous reports week post vaccination to record peak titers by the 12 [28,30,33,40] who mentioned that vaccination of week post vaccination for both types. These antibody calves with the locally produced bivalent FMD vaccine titers remained within the protective level up to 36 adjuvanted with Montanide ISA 206 induced higher weeks post vaccination in the 2 farms under study.…”