“…Previous studies have demonstrated that prolonged intra-and inter-atrial conduction times and inhomogeneous propagation of sinus impulses would be the characteristics of increased AF risk in various clinical conditions during sinus rhythm. 27 It has been studied in some clinical conditions, [28][29][30][31] and it was found that AEMD is significantly correlated with the prolonged maximum P-wave duration, P-wave dispersion, and degree of histopathological changes. [32][33][34] Previous studies evaluated the predictive role of intra-left atrial electromechanical delay for paroxysmal atrial fibrillation recurrence after cardioversion, [35][36][37] lone atrial fibrillation attack, 38 new-onset AF after acute myocardial infarction, 39 occurrence of atrial arrhythmias in adult patients with congenital heart diseases, 40 myotonic dystrophy type 1, 41 and atrial septal aneurysm.…”