клинический случай clinical case F ournier gangrene (epifascial necrosis) is a specific form of necrotizing fasciitis that affects patients of both sexes and of any age. However, it is mainly observed in men suffering from immunodeficiency [1, 2], especially in those with injuries of the genitals and perineum. Cases of the development of Fournier gangrene after piercing and injection of drugs into the veins of the penis and cavernous bodies have been described [3, 4]. Streptococcus, Staphylococcus, Escherichia coli, and microbial associations involving anaerobic bacteria play a significant role as infectious agents [5-7]. The disease is characterized by the vastness and high rate of lesion spread; total necrosis can develop up to the anterior abdominal wall and even further into the axillary areas as well as the inner surface of the thighs. This is owing to the mixed nature of the infection, involving the release of enzymes and toxins by microbes; this causes high pathogenic-ity and thrombosis in the vessels, resulting in tissue necrosis. Owing to tissue hypoxia, anaerobes that produce hyaluronidase, lecithinase, and collagenase begin to actively multiply. These favorable conditions aid the microorganisms in rapidly overcoming interstitial and fascial barriers [8, 9]. The mortality rate is extremely high and can exceed 40 % [10]. Nevertheless, successful treatment of these patients has been previously reported [11-14].Reports of Fournier gangrene in women are rare [15], and there are no data in obstetric practice. Pregnancy itself, particularly its pathological course, contributes to the development of immunodeficiency [16,17]. Surgical interventions on the perineum (i.e., episiotomies, vaginal tissues, and perineum restoration after traumatic childbirth); surgical delivery, including cesarean section; and damage to nearby organs, specifically the urethra, bladder, and rectum, all contribute to the parturient occurrence of this disease.