Exposure assessment for a population-based case-control study combining a job-exposure matrix with interview data

Semple, Sean; Dick, Finlay; Cherrie, John W.

doi:10.5271/sjweh.785

Cited by 49 publications

(35 citation statements)

References 27 publications

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“…Parents in the present study provided a job title, but no information regarding chemical exposure, control measures or the tasks performed at work. The study coded the job titles and constructed a JEM to the main classes of known occupational asthmogens [22] using previous experience in other population-based occupational/ health studies [18]. With all JEMs, there is misclassification of exposure because of variability in the intensity of exposure within jobs [23], and the present classification methods are likely to be similarly limited.…”

Section: Discussionmentioning

confidence: 99%

“…For each of the 353 SOC2000 codes, two experienced occupational hygienists with experience of retrospective exposure assessment [18] independently derived semi-quantitative ratings (high, medium, low and zero) for the intensity of workplace and take-home exposure to 11 major occupational sensitisers (wood, diisocyanate, flour, glues/ resins, animals, solder, enzymes, biocides/fungicides, foods, natural rubber latex and dyes). Assessment of the intensity of workplace exposure was based on typical UK working conditions during the early 1990s and took account of control measures, personal protective equipment and ventilation/ extraction methods in common use within that sector.…”

Section: Parental Occupationmentioning

confidence: 99%

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Parental occupation is a risk factor for childhood wheeze and asthma

Tagiyeva

Devereux²,

Semple³

et al. 2009

Eur Respir J

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The present birth cohort study investigated whether or not childhood wheeze and asthma are associated with parental exposure to occupational sensitisers that cause asthma.Parental occupation, from the Avon Longitudinal Study of Parents and Children (ALSPAC), was related to wheeze, asthma, ventilatory function, airway responsiveness and atopic sensitisation in children aged 0-102 months.Occupation was recorded for 11,193 mothers and 9,473 fathers antenatally, and for 4,631 mothers and 5,315 fathers post-natally. Childhood respiratory outcomes were not associated with parental occupational exposure to diisocyanates, glues/resins, dyes, animal dust, solder, enzymes and wood dust. Maternal post-natal occupational exposure to latex and/or biocides/fungicides increased the likelihood of childhood wheeze and asthma. High levels of latex or biocide/fungicide exposure were associated with an OR (95% CI) of 1.26 (1.07-1.50) and 1.22 (1.02-2.05), respectively, for wheezing up to 81 months. Combined maternal latex and biocide/fungicide exposure increased the likelihood of childhood wheeze (1.22 (1.03-1.43)) and asthma. High paternal occupational flour dust exposure was associated with an increased likelihood of wheeze after 30 months (2.31 (1.05-5.10)) and asthma by 91 months (3.23 (1.34-7.79)).Maternal occupational exposure to latex and/or biocides and paternal exposure to flour dust increases the risk of childhood asthma. Further studies in this area are justified.

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Section: Discussionmentioning

confidence: 99%

Section: Parental Occupationmentioning

confidence: 99%

Parental occupation is a risk factor for childhood wheeze and asthma

Tagiyeva

Devereux²,

Semple³

et al. 2009

Eur Respir J

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“…In the present study the mass transfer of soil-sorbed hydrophobic pesticides from the soil phase to the solution phase was achieved by introducing a bacterial population capable of solubilizing the pesticide through biosurfactant production that subsequently degrades the pesticide. There are many reports on the enhanced bioavailability of organic contaminants with low water solubility by using biosurfactants and biosurfactant producing microbes (Paria, 2008;Preethy and Nilanjana, 2010;Jing et al, 2010), but there are few studies on the enhanced bioavailability of pesticides in water and soil by adding a biosurfactant externally (Natesan et al, 2012;Semple et al, 2004;Mata-Sandoval et al, 2000;Arunkumar et al, 2011;Kosaric, 2001;Wattanaphon and Kerdsin, 2008). Natesan et al (2012) reported 30e50% enhanced degradation of freshly spiked HCH isomers (200 mg/g) Sphingomonas sp.…”

Section: Biodegradation Of Endosulfan and Other Pesticides In Differementioning

confidence: 96%

Residues of endosulfan in surface and subsurface agricultural soil and its bioremediation

Odukkathil

Vasudevan

2016

Journal of Environmental Management

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“…To assess the relationship between occupational exposure and risk of lung damage an occupational hygienist created a job exposure matrix [15] using Standard Occupational Classifications 2000, generated using the computer assisted structured coding tool [16] system. Job codes were divided into three groups according to the likelihood and probable intensity of exposure to agents known to cause occupational lung disease.…”

Section: Clinical Phenotypingmentioning

confidence: 99%

Outdoor air pollution is associated with disease severity in α₁-antitrypsin deficiency

Wood

Harrison²,

Semple³

et al. 2009

Eur Respir J

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The aim of this study was to determine whether long-term air pollution exposure is associated with clinical phenotype in a 1 -antitrypsin deficiency.In total, 304 PiZZ subjects underwent full lung function testing and quantitative high-resolution computed tomography to identify the presence and severity of the disease. Mean annual air pollutant data for 2006 was matched to the location of patients' houses and used in regression models to identify phenotypic associations with pollution, controlling for covariates. Relative trends in pollution levels were assessed to validate use of a single year's data to indicate longterm exposure.Pollutant levels correlated significantly with one another, with higher levels of primary particles, SO 2 and NO 2 being associated with lower ozone levels. Regression models showed that estimated higher exposure to ozone was associated with worse gas transfer and more severe emphysema. Regression parameters suggested that significance from other pollutants was due to collinearity with ozone. The 2006 pollutant levels showed linear relationships with cumulative years, thus validating the model.Higher exposures to ozone may be associated with worse respiratory status in a 1 -antitrypsin deficiency, identifying a group susceptible to ambient air pollution.

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Exposure assessment for a population-based case-control study combining a job-exposure matrix with interview data

Cited by 49 publications

References 27 publications

Parental occupation is a risk factor for childhood wheeze and asthma

Parental occupation is a risk factor for childhood wheeze and asthma

Residues of endosulfan in surface and subsurface agricultural soil and its bioremediation

Outdoor air pollution is associated with disease severity in α₁-antitrypsin deficiency

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Exposure assessment for a population-based case-control study combining a job-exposure matrix with interview data

Cited by 49 publications

References 27 publications

Parental occupation is a risk factor for childhood wheeze and asthma

Parental occupation is a risk factor for childhood wheeze and asthma

Residues of endosulfan in surface and subsurface agricultural soil and its bioremediation

Outdoor air pollution is associated with disease severity in α1-antitrypsin deficiency

Contact Info

Product

Resources

About

Outdoor air pollution is associated with disease severity in α₁-antitrypsin deficiency