Exposure to phagolysosomal simulated fluid altered the cytotoxicity of PET micro(nano)plastics to human lung epithelial cells

Baysal, Asli; Soyocak, Ahu; Saygin, Hasan; Saridag, Ayse Mine

doi:10.1080/15376516.2023.2256847

Toxicology Mechanisms and Methods

2023

DOI: 10.1080/15376516.2023.2256847

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Exposure to phagolysosomal simulated fluid altered the cytotoxicity of PET micro(nano)plastics to human lung epithelial cells

Asli Baysal,

Ahu Soyocak,

Hasan Saygin

et al.

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2024

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Cited by 4 publications

References 96 publications

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Oxidative stress, biofilm-formation and activity responses of P. aeruginosa to microplastic-treated sediments: Effect of temperature and sediment type

Saygin,

Tilkili,

Kayisoglu

et al. 2024

Environmental Research

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Oxidative stress, biofilm-formation and activity responses of P. aeruginosa to microplastic-treated sediments: Effect of temperature and sediment type

Saygin,

Tilkili,

Kayisoglu

et al. 2024

Environmental Research

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Year-long and seasonal differences of PM _2.5 chemical characteristics and their role in the viability of human lung epithelial cells (A549)

Baysal,

Saygin,

Soyocak

et al. 2024

Journal of Environmental Science and Health, Part A

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Airborne micro- and nanoplastics: emerging causes of respiratory diseases

Gou,

Wu,

et al. 2024

Part Fibre Toxicol

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Airborne micro- and nanoplastics (AMNPs) are ubiquitously present in human living environments and pose significant threats to respiratory health. Currently, much research has been conducted on the relationship between micro- and nanoplastics (MNPs) and cardiovascular and gastrointestinal diseases, yet there is a clear lack of understanding regarding the link between AMNPs and respiratory diseases. Therefore, it is imperative to explore the relationship between the two. Recent extensive studies by numerous scholars on the characteristics of AMNPs and their relationship with respiratory diseases have robustly demonstrated that AMNPs from various sources significantly influence the onset and progression of respiratory conditions. Thus, investigating the intrinsic mechanisms involved and finding necessary preventive and therapeutic measures are crucial. In this review, we primarily describe the fundamental characteristics of AMNPs, their impact on the respiratory system, and the intrinsic toxic mechanisms that facilitate disease development. It is hoped that this article will provide new insights for further research and contribute to the advancement of human health. Graphical Abstract

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Exposure to phagolysosomal simulated fluid altered the cytotoxicity of PET micro(nano)plastics to human lung epithelial cells

Cited by 4 publications

References 96 publications

Oxidative stress, biofilm-formation and activity responses of P. aeruginosa to microplastic-treated sediments: Effect of temperature and sediment type

Oxidative stress, biofilm-formation and activity responses of P. aeruginosa to microplastic-treated sediments: Effect of temperature and sediment type

Year-long and seasonal differences of PM _2.5 chemical characteristics and their role in the viability of human lung epithelial cells (A549)

Airborne micro- and nanoplastics: emerging causes of respiratory diseases

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Exposure to phagolysosomal simulated fluid altered the cytotoxicity of PET micro(nano)plastics to human lung epithelial cells

Cited by 4 publications

References 96 publications

Oxidative stress, biofilm-formation and activity responses of P. aeruginosa to microplastic-treated sediments: Effect of temperature and sediment type

Oxidative stress, biofilm-formation and activity responses of P. aeruginosa to microplastic-treated sediments: Effect of temperature and sediment type

Year-long and seasonal differences of PM 2.5 chemical characteristics and their role in the viability of human lung epithelial cells (A549)

Airborne micro- and nanoplastics: emerging causes of respiratory diseases

Contact Info

Product

Resources

About

Year-long and seasonal differences of PM _2.5 chemical characteristics and their role in the viability of human lung epithelial cells (A549)