Impingement is a clinical scenario of painful functional limitation of the shoulder, 1 thought to be secondary to compression or altered dynamics that irritate and ultimately damage the tissues around the shoulder joint. Shoulder impingement is currently subdivided into external (subacromial) and internal impingement. External impingement is further subdivided into primary and secondary, and internal impingement into posterosuperior and anterosuperior (►Fig. 1). 2-8
External ImpingementThe theory of external impingement syndrome was first proposed by Neer in 1972 to describe shoulder pain associated with varying degrees of chronic bursitis and partial-or full-thickness tears of the rotator cuff. 7 However, this was before the advent of modern imaging, primarily ultrasound (US) and MR imaging. External/subacromial impingement occurs when the supraspinatus tendon and subacromialsubdeltoid (SASD) bursa are impinged between the humeral head, the acromion, and coracoacromial ligament. Early theories on etiology focused on anatomical abnormalities of the coracoacromial arch (►Fig. 2). However, there is growing evidence that scapular dysfunction may be more significant, especially in the patient <40 years of age.
Etiology of ImpingementAnatomical abnormalities of the arch have highlighted morphological variation in the acromion and acromioclavicular (AC) joint. The morphology of the acromion has been categorized into three types (type I flat, type II concave, and type III hooked). It has been suggested that the hooked type III configuration may predispose to external impingement. 9 However, it is more likely that (unless the anatomical changes are gross) acromial changes are secondary rather than primary. Anterior and lateral downsloping of the acromion is also implicated in external impingement, particularly in tears of the supraspinatus tendon at its attachment. Acromioclavicular joint arthrosis, with osteophytes on the undersurface of the acromium, 1 and os acromiale are both believed to be causative in external impingement but are not accurately assessed with US and require subacromial outlet radiographs or MR imaging for optimal visualization.Functional abnormalities lead to external impingement by causing a relative decrease in the subacromial space due to glenohumeral instability or abnormal scapulothoracic movement. In other words, it is proximal displacement of the humeral head that causes impingement of the subacromial space against the coracoacromial arch rather than the reverse. These are encompassed within the term the SICK scapula syndrome (scapular malposition, inferomedial prominence, coracoid pain, scapular dyskinesis). 9a The disturbed balance of forces between the rotator cuff muscles and the deltoid muscle leads to elevation of the humeral head and secondary impingement of the contents of the subacromial space on the coracoacromial arch. Abnormal shoulder biomechanics also contribute to glenohumeral instability, particularly the
AbstractThis update examines recent articles and evidence for the role ...