2009
DOI: 10.1002/clc.20449
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Factors to Predict Recurrence of Atrial Fibrillation in Patients with Hypertension

Abstract: Backgound: Patients with essential hypertension had a higher recurrence rate of atrial fibrillation (AF) than patients without hypertension. This study evaluated the role of various parameters for predicting recurrence after conversion of AF in patients with essential hypertension. Methods: We retrospectively analyzed 136 patients with essential hypertension and a known history of AF. P-wave duration and dispersion were measured in a 12-lead electrocardiograph. The size and function of the left atrium (LA) and… Show more

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Cited by 16 publications
(10 citation statements)
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“…PD has been studied in some other conditions such as hypertension [29], obesity [30], diabetes mellitus [31], metabolic syndrome [32], dilated cardiomyopathy [33], myocardial infarction [34], atrial septal defect [35], hypertrophic cardiomyopathy [36], obstructive sleep apnea [37], Emery Dreifuss muscular dystrophy [38] and Wilson's disease [39]. The exact mechanism of PD prolongation in these clinical conditions is not well known, but it is thought that structural and electrophysiological changes in the atrial myocardium caused by elevated plasma volume [40], ventricular diastolic dysfunction [41] and enhanced neurohormonal activation [42], typical conditions of these deseases, may contribute to left atrial enlargement and electrical instability.…”
Section: Discussionmentioning
confidence: 99%
“…PD has been studied in some other conditions such as hypertension [29], obesity [30], diabetes mellitus [31], metabolic syndrome [32], dilated cardiomyopathy [33], myocardial infarction [34], atrial septal defect [35], hypertrophic cardiomyopathy [36], obstructive sleep apnea [37], Emery Dreifuss muscular dystrophy [38] and Wilson's disease [39]. The exact mechanism of PD prolongation in these clinical conditions is not well known, but it is thought that structural and electrophysiological changes in the atrial myocardium caused by elevated plasma volume [40], ventricular diastolic dysfunction [41] and enhanced neurohormonal activation [42], typical conditions of these deseases, may contribute to left atrial enlargement and electrical instability.…”
Section: Discussionmentioning
confidence: 99%
“…PD is a noninvasive indicator of intra-atrial conduction heterogeneity producing substrate for reentry, which is a pathophysiological mechanism of atrial fibrillation [16,17]. PD has been studied in some other conditions such as hypertension [18], obesity [19], diabetes mellitus [20], metabolic syndrome [21], dilated cardiomyopathy [22], myocardial infarction [23], atrial septal defect [24], hypertrophic cardiomyopathy [25], obstructive sleep apnea [26] and Wilson's disease [27]. The exact mechanism of PD prolongation in these clinical conditions is not well known, but it is thought that structural and electrophysiological changes in the atrial myocardium caused by elevated plasma volume [28], ventricular diastolic dysfunction [29] and enhanced neurohormonal activation [30], that accompany these diseases, may contribute to left atrial enlargement and electrical instability.…”
Section: Discussionmentioning
confidence: 99%
“…The LA maximum volume (V max ) at the end‐systolic phase, LA minimum volume (V min ) at the end‐diastolic phase, and LA volume before atrial systole (V p ) (origin of P‐wave in electrocardiography) were measured, and all volumes were indexed to body surface area (BSA) and expressed in mL/m 2 . Parameters of LA function were calculated as follows: LA passive emptying volume (LAPEV): V max − V p ; LA passive emptying fraction (LAPEF): [(V max − V p )/V max ] × 100; LA active emptying volume (LAAEV): V p − V min ; LA active emptying fraction (LAAEF): [(V p − V min )/V p ] × 100; LA total emptying volume (LATEV): V max − V min ; LA ejection fraction (LAEF): [(V max − V min )/V max ] × 100 …”
Section: Methodsmentioning
confidence: 99%