Fetal ascites associated with ABO incompatibility: Case report and review of the literature

Stiller, Robert J.; Herzlinger, Robert; Siegel, Stacey; Whetham, John

doi:10.1016/s0002-9378(96)70057-1

Cited by 20 publications

(16 citation statements)

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“…There was no evidence for congenital infection, including malaria, the G6PD was normal, red cell morphology returned to normal at a few weeks of age so excluding a red cell membrane disorder and the high HbF (91·8%) and clinical presentation made significant neonatal sickling highly unlikely. An initial negative DAT does not exclude the diagnosis as it is well established that ABO haemolytic disease can be associated with a negative or weakly reactive DAT in the presence of severe haemolysis (Sherer et al ., 1991; Stiller et al ., 1996).…”

Section: Discussionmentioning

confidence: 99%

“…1 in 150 births is complicated by mild ABO HDN, characterized by jaundice without anaemia, whereas severe haemolysis will be seen in only 1 in 3000 neonates. It is rarer still for feto‐maternal ABO incompatibility to impact on fetal life with only six case reports to date of hydrops fetalis secondary to ABO incompatibility (Miller & Petrie, 1963; Gilja & Shah, 1988; Sherer et al ., 1991; Stiller et al ., 1996; McDonnell et al ., 1998). None of these case reports involved the use of intra uterine transfusions, and we could find no previous reports of both fetuses in a twin pregnancy being affected.…”

Section: Discussionmentioning

confidence: 99%

“…It is of additional interest that in all three of our cases the infants were B Rh D positive. In four of the six previously published cases, the neonates were also group B (Sherer et al ., 1991; Stiller et al ., 1996; McDonnell et al ., 1998).…”

Section: Discussionmentioning

confidence: 99%

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ABO incompatibility due to immunoglobulin G anti‐B antibodies presenting with severe fetal anaemia

Ziprin

Payne

Hamidi

et al. 2005

Transfusion Medicine

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ABO incompatibility is a common haematological problem affecting the newborn. The haemolysis is widely accepted to follow a relatively benign course rarely causing the escalating levels of hyperbilirubinaemia and significant anaemia associated with Rh haemolytic disease of the newborn. Case reports of fetal hydrops secondary to ABO incompatibility are particularly rare. We describe two cases, first that of a twin pregnancy with both fetuses developing severe anaemia at 20 weeks gestation, and then a second case of a preterm baby demonstrating aggressive haemolysis and anaemia within hours of delivery. Both mothers were of black Africian origin and both were identified to have elevated titres of IgG anti-B antibodies.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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ABO incompatibility due to immunoglobulin G anti‐B antibodies presenting with severe fetal anaemia

Ziprin

Payne

Hamidi

et al. 2005

Transfusion Medicine

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“…Although fetal-maternal ABO incompatibility is considered rarely causing severe fetal diseases in some countries [27], severe ABO-HDFN has actually happened in a small population [2,28,29]. Our protocol could be used to determine fetal ABO blood groups in middle pregnancy, which is helpful for predicting and diagnosing ABO-HDFN.…”

Section: Discussionmentioning

confidence: 99%

“…It has been reported that mild ABO-HDFN occurs in about 1 of 150 births and the newborns with mild ABO-HDFN are commonly treated by phototherapy. The severe ABO-HDFN occurs in about 1 of 3000 births and the newborns with severe ABO-HDFN may need exchange transfusion like those with severe RhD-HDFN [2]. At present, pregnant women usually take prenatal serological tests of blood group and red cell antibody to predict HDFN.…”

Section: Introductionmentioning

confidence: 99%