Five-chambered heart: a 20-year story of left atrial appendage aneurysm

Left Atrial Appendage Aneurysm: A Systematic Review of 82 Cases

“…52 Surgical or endoscopic resection eliminates the risk of both thromboembolism and tachyarrhythmia. 7,8,71,72 Limitations of the Study: Several limitations of our study should be noted. Hence, in patients with large LAAA and those with associated atrial fibrillation and/or biatrial enlargement concurrent Cox-Maze III type or other ablative procedure at the time of LAAA resection should be performed.…”

“…In those not undergoing surgery due to various reasons medical management should be directed to treat atrial tachyarrhythmia and thromboembolic complication. 7,8,71,72 Limitations of the Study: Several limitations of our study should be noted. This is a retrospective series of reported cases in the current literature, and has inherent biases related to such studies including selection and publication biases.…”

Incidentally detected congenital giant left atrial appendage aneurysm in a child: MRI findings

“…Angiography is also an invasive procedure, which should be reserved for patients with concomitant cardiac anomalies or for cases in which echocardiography or MRI are inconclusive. Cardiac multidetector CT is valuable for evaluating the anatomy of the coronary artery when compression of the left coronary artery or its branches is suspected (4,5).…”

Congenital giant left atrial appendage aneurysm: a case report

“…Plonska-Gosciniak et al reported a case of a 45-year-old patient with a giant LAAA (112 × 49 × 80 mm) who presented with exercise intolerance and palpitation and who refused surgical resection but accepted drug management. The patient remained stable with occasional supraventricular arrhythmia at the 20-year follow-up [2]. Sharma et al reported a case of a 35-year-old patient with LAAA (51 × 66 mm) who declined surgery; the patient’s shortness of breath had resolved at the 6-month follow-up with oral anticoagulation treatment, alone [3].…”

“…Despite the congenital cause, clinical symptoms usually do not arise until the second to third decades of life. Some LAAA cases are detected incidentally, but most are diagnosed when patients present with palpitation, dyspnea, angina, or thromboembolic events [2–4]. Because LAAA predisposes to thromboembolism, arrhythmias, and heart failure, surgical resection is generally recommended [5].…”