Focal cerebral deficits in severe hypomagnesemia

Leicher, Carol R.; Mezoff, Adam; Hyams, Jeffrey S.

doi:10.1016/0887-8994(91)90070-2

Cited by 26 publications

(8 citation statements)

References 6 publications

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“…But patients with hypomagnesaemia may also present many further neurological symptoms such as seizures, paraesthesias, myoclonus, fasciculations, ataxia, dysarthria, irritability, disorientation, psychosis, vertigo and confusion 3. Recurrent symptomatology4 and even focal cerebral deficits such as hemiparesis5 has been described.…”

Section: Discussionmentioning

confidence: 99%

Recurrent confusional episodes associated with hypomagnesaemia due to esomeprazol

Delgado¹,

Calleja

Suárez

et al. 2013

BMJ Case Reports

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SUMMARYIn February 2011, the Food and Drug Administration informed that prescription of proton pump inhibitor (PPI) drugs may cause low serum magnesium levels if taken for prolonged periods of time. We present an ex-smoker, 76-year-old man, with high blood pressure, diabetes mellitus and Barrett's oesophagus (treated with esomeprazole since 2003) admitted due to fluctuating aphasia. Neurovascular and neuroimaging studies were normal. Dyslipidemia and atrial arrhythmia were discovered. The patient was discharged with the diagnosis of left middle cerebral artery transient ischaemic attack and anticoagulation treatment was recommended. The patient returned to the emergency department on further two occasions (confusional episodes) and was admitted in order to complete the neurological study that was normal. The patient was discharged with the diagnosis of probable epileptic seizures. After a week, he was admitted due to generalised temblor and unsteadiness. A complete blood test was performed and showed a severe hypomagnesaemia (not previously performed). BACKGROUND

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Section: Discussionmentioning

confidence: 99%

Recurrent confusional episodes associated with hypomagnesaemia due to esomeprazol

Delgado¹,

Calleja

Suárez

et al. 2013

BMJ Case Reports

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show abstract

“…Although they are unlikely to occur without concomitant mental status alteration, hypocalcemic seizures may be focal in nature and mimic a monoparesis [12]. Similarly, severe hypomagnesemia (b0.8 mEq/L) occasionally presents with focal neurologic deficits [13]. When the results of blood glucose and electrolyte determinations are not immediately available, administration of aliquots of naloxone and/or flumazenil may be considered in the interim to antagonize the central effects of administered opioids or benzodiazepines, but must be weighed against potential adverse effects of these drugs.…”

Section: Discussionmentioning

confidence: 96%

Transient anesthetic-induced worsening of existing left-sided weakness in a patient undergoing elective anterior cervical discectomy

Joffe

Ahmed

Trost

et al. 2009

Journal of Clinical Anesthesia

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“…Hypomagnesemia is associated with multiple neurological abnormalities, including irritability, seizures, paralysis, and choreiform movements [36,37]. The trace element manganese is routinely administered intravenously to TPN patients at a dosage of 100 to 800 μg/d.…”

Section: Indicationsmentioning

confidence: 99%