Foix-chavany syndrome: CT study and clinical report of three cases

Abstract

The Foix-Chavany syndrome is a neurological entity characterized by linguo-bucco-facial apraxia almost always caused by disturbed cerebral circulation. Three typical cases of this syndrome are described and the role of the CT scan to obtain a definite diagnosis is emphasized.

Cortical innervation of the hypoglossal nucleus in the non‐human primate (Macaca mulatta)

“…Our work also suggests that spared cortical areas on the dorsolateral surface (LPMCd,) as well as medial surface (M2, M3, M4 and the anterior cingulate gyrus) may support tongue motor recovery following unilateral cortical stroke, which needs to be assessed in future patient studies. Indeed, if present in the human brain, our findings of medial wall corticohypoglossal projections may be of particular importance for favorable recovery of tongue movement control in patients with a history of MCA strokes that initially affect one hemisphere, followed by a second stroke affecting the other hemisphere (e.g., Umapathi et al, 2000), or in patients sustaining bilateral opercular MCA occlusion (e.g., Ferrari et al, 1979; Grattan-Smith et al, 1989; Nisipeanu et al, 1997; Suresh and Deepa, 2004)…”

Foix-Chavany-Marie-syndrome — neurological, neuropsychological, CT, MRI, and SPECT findings in a case progressive for more than 10 years

“…CT shows more or less symmetrical bilateral infarctions (Mariani et al 1980;Colombo et al 1983;Villa et al 1984;Levine and Mohr 1979;Ferrari et al 1979;dalla Pria et al 1979) or tissue loss with marked brain atrophy Sandyk and Brennan 1983). This paper reports the case of a patient presenting with a primary bilateral opercular syndrome that has been slowly progressing for over 10 years.…”

“…Several varieties of Foix-Chavany-Marie (FCM) syndrome have been described: accompanied by corn-* Supported by the Hirnliga, Heidelberg, Federal Republic of Germany Offprint requests to: C. Lang, Department of Neurology, University of Erlangen-Nfirnberg, Schwabachanlage 6, D-8520 Erlangen, Federal Republic of Germany plete speechlessness (speech suppression) without aphasia (Alajouanine and Thurel 1933;Arnould et al 1967;Cappa et al 1987;Colombo et al 1983;Mariani et al 1980;Villa and Caltagirone 1984), by aphasia and/or oral apraxia (Colombo et al 1983;Ferrari et al 1979;Levine and Mohr 1979;Mariani et al 1980;Sandyk and Brennan 1983;, and by pure word deafness (dalla Pria et al 1979). Depending on the relative involvement of the anterior (Biller et al 1981) or the posterior operculum, two varieties of the syndrome are distinguished.…”

Neural substrates of the ‘low‐level’ system for speech articulation: Evidence from primary opercular syndrome

“…The syndrome is associated with bilateral, cortical/subcortical lesions of the anterior part of the frontal operculum. The acute form, which is due to bilateral, acquired lesions, is the most common one (Bursaw & Duginski, ; Ferrari, Boninsegna, & Beltramello, ; Mao, Coull, Golper, & Rau, ; Mariani, Spinnler, Sterzi, & Vallar, ; Praveen‐Kumar & Pramod, ; Villa & Caltagirone, ). Less frequently monolateral lesions have been reported (Nitta, Shiino, Sakaue, & Nozaki, ).…”