Food and Drug Administration Petition on Food Labeling: An Update From the American Dietetic Association and National Kidney Foundation

Karalis, Maria

doi:10.1053/j.jrn.2007.08.011

Cited by 9 publications

(8 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…So, limiting consumption of inorganic phosphate additives is a strategic way to decrease phosphorus intake without affecting protein intake (77), which is particularly important for patients with CKD on dialysis. At minimum, inclusion of phosphorus content on food labels, and the quantification of phosphorus from inorganic phosphate food additives would be helpful for those seeking to limit their phosphorus intake (4, 7, 15, 17, 63, 78–80). …”

Section: Discussionmentioning

confidence: 99%

Effects of Excessive Dietary Phosphorus Intake on Bone Health

Vorland

Stremke

Moorthi

et al. 2017

Curr Osteoporos Rep

View full text Add to dashboard Cite

Purpose of Review The purpose of this review is to provide an overview of dietary phosphorus, its sources, recommended intakes, and its absorption and metabolism in health and in chronic kidney disease, and to discuss recent findings in this area with a focus on the effects of inorganic phosphate additives in bone health. Recent Findings Recent findings show that increasing dietary phosphorus through inorganic phosphate additives has detrimental effects on bone and mineral metabolism in humans and animals. There is new data supporting an educational intervention to limit phosphate additives in patients with chronic kidney disease to control serum phosphate. Summary The average intake of phosphorus in the US well-above the recommended dietary allowance. Inorganic phosphate additives, which are absorbed at a high rate, account for a substantial and likely underestimated portion of this excessive intake. These additives have negative effects on bone metabolism, and present a prime opportunity to lower total phosphorus intake in the U.S. Further evidence is needed to confirm whether lowering dietary phosphorus intake would have beneficial effects to improve fracture risk.

show abstract

Section: Discussionmentioning

confidence: 99%

Effects of Excessive Dietary Phosphorus Intake on Bone Health

Vorland

Stremke

Moorthi

et al. 2017

Curr Osteoporos Rep

View full text Add to dashboard Cite

show abstract

“…Total phosphorus intake is not only dependent on the raw quantities found in “natural” protein sources, but also on the growing amount of phosphorus-rich additives in processed and fast foods 9, 10, 19, 21. These additives can augment phosphorus intake by as much as 1000 mg per day 13 but are often not captured by standard dietary instruments because there are no requirements for the food industry to quantify their amount in product labeling 35, 36. As such, there can be large gaps between estimated phosphorus intake vs. actual phosphorus intake 9, 19, 37.…”

Section: Discussionmentioning

confidence: 99%

Impact of Poverty on Serum Phosphate Concentrations in the Third National Health and Nutrition Examination Survey

Gutiérrez

Isakova

Enfield

et al. 2011

Journal of Renal Nutrition

View full text Add to dashboard Cite

Objective-Increased serum phosphate is associated with adverse health outcomes. High intake of inexpensive processed and fast foods is common in impoverished communities and is linked with excessive dietary phosphorus intake and elevated serum phosphate concentrations in chronic kidney disease patients. We examined the impact of socioeconomic status on dietary phosphorus intake and serum phosphate concentrations in the general population.Design-Cross-sectional study.Participants-14,261 adult participants in the Third National Health and Nutrition Examination Survey.Predictors and Outcomes-Poverty to income ratio (PIR; family income indexed to the federal poverty level) was the primary index of socioeconomic status. Serum phosphate was the primary outcome variable.Results-Although estimated phosphorus intake decreased with decreasing quartiles of PIR (P < 0.001), serum phosphate was inversely associated with PIR (P = 0.003). The relationship between lower PIR and higher serum phosphate remained significant after adjustment for demographic, laboratory, and dietary intake characteristics (P = 0.02). Compared to participants in the highest PIR quartile (income >300% of the federal poverty level), participants in the lowest quartile (income < the federal poverty level) had more than twice the odds of hyperphosphatemia (≥4.4 mg/dl) in unadjusted and multivariable-adjusted logistic regression analyses (OR 2.2, 95%CI 1.5, 3.2).Conclusions-Although lower income was associated with decreased estimated phosphorus intake, increasing poverty was independently linked with increased serum phosphate and higher likelihood of hyperphosphatemia. These findings may indicate that conventional dietary

show abstract

“…Thus, total phosphorus intake depends not only on the raw quantities found in “natural” protein sources, but also on the growing amount of phosphorus‐rich additives in processed and fast foods as well as in beverages 19–22 . These additives can augment phosphorus intake by as much as 1,000 mg per day, but they are often not captured by standard dietary instruments because the amount of phosphorous in foods and beverages does not presently need to be quantified on product labels 23,24 . Consequently, large gaps may exist between estimated and actual phosphorus intake 19,21,25 …”

Section: Levels Of Phosphorus Intakementioning

confidence: 99%

Dietary phosphorus in bone health and quality of life

et al. 2012

View full text Add to dashboard Cite

Awareness of phosphorus intake is important because both phosphorus deficiency and overloading impair bone health and quality of life. Phosphorus consumption is increasing in many countries. Most dietary phosphorus is contained in protein-rich foods such as meat, milk, cheese, poultry, fish, and processed foods that contain phosphate-based additives to improve their consistency and appearance. Elevation of extracellular phosphorus levels causes endothelial dysfunction and medial calcification, which are closely associated with the development of cardiovascular disease (CVD). Long-term excessive phosphorus loading, even if it does not cause hyperphosphatemia, can be a risk factor for CVD. In epidemiological studies, higher levels of phosphorus intake have been associated with reduced blood pressure. Interestingly, when examined further, phosphorus from dairy products, but not from other sources, was usually associated with lower blood pressure. A dietary approach to phosphorus reduction is particularly important to prevent bone impairment and CVD in patients with chronic kidney disease. In order to improve bone health and quality of life in the general population, the impact of phosphorous, including in processed foods, should be considered, and measures to indicate the amount of phosphorous in food products should be implemented.

show abstract

Food and Drug Administration Petition on Food Labeling: An Update From the American Dietetic Association and National Kidney Foundation

Cited by 9 publications

References 0 publications

Effects of Excessive Dietary Phosphorus Intake on Bone Health

Effects of Excessive Dietary Phosphorus Intake on Bone Health

Impact of Poverty on Serum Phosphate Concentrations in the Third National Health and Nutrition Examination Survey

Dietary phosphorus in bone health and quality of life

Contact Info

Product

Resources

About