Fr�hstadien der experimentellen Herzhypertrophie im elektronenmikroskopischen Bild

Onishi, Shinsuke; Zittel, R. X.

doi:10.1007/bf00660146

Cited by 8 publications

(2 citation statements)

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“…Most publications deal with early stages of cardiac hypertrophy (1,3,7, 11,12,16,17,23,24,28) but only few studies refer to its prospective regression after load suppression. Most publications deal with early stages of cardiac hypertrophy (1,3,7, 11,12,16,17,23,24,28) but only few studies refer to its prospective regression after load suppression.…”

Section: Discussionmentioning

confidence: 99%

“…Most publications deal with early stages of cardiac hypertrophy (1,3,7, 11,12,16,17,23,24,28) but only few studies refer to its prospective regression after load suppression.According to Beznak et al (4), however, hypertrophy of the heart is readily reversible, regardless of its origin (pressure load, hyperthyroidism, anemia or DOCA salt hypertension) or its duration. This enlargement may be partly due to intracellular edema, characteristic of cell damage.…”

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confidence: 99%

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Entwicklung und Rückbildung druckinduzierter Herzhypertrophie

et al. 1978

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Light and electron microscopy, combined with morphometry, were utilized to study myocardial cell modifications induced by a temporary abdominal aortic constriction in the rat. During the early stage of active hypertrophy, cell enlargement in the subendocardial layers of the left ventricle was predominant. This enlargement may be partly due to intracellular edema, characteristic of cell damage. Degenerative foci leading to fibrous scars were found primarily in the subendocardium and midwall layers. The times taken by the different cell structures to adapt to modifications of the load were rather different; some adapted very rapidly (nucleoli, intercalated discs), while others were much slower (cell diameter, mitochondria).

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Section: Discussionmentioning

confidence: 99%

“…Most publications deal with early stages of cardiac hypertrophy (1,3,7, 11,12,16,17,23,24,28) but only few studies refer to its prospective regression after load suppression.According to Beznak et al (4), however, hypertrophy of the heart is readily reversible, regardless of its origin (pressure load, hyperthyroidism, anemia or DOCA salt hypertension) or its duration. This enlargement may be partly due to intracellular edema, characteristic of cell damage.…”

mentioning

confidence: 99%