Abstract:BackgroundExperimental SLE-models show that complement activation may be a key part of lupus cerebritis and lupus-thrombosis. It is also known that complement factors contribute to the pathology of inflammatory central nervous system (CNS) and neurodegenerative diseasesObjectivesTo analyse serum levels of anti-C1q, C1q circulating immune complexes (CIC), complement activation and complement components in systemic lupus erythematosus (SLE) patients during the first CNS neuropsychiatric (NP) event and to define … Show more
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