“…Such controls may reduce the value of the basic reproduction number related to the case of absence of controls, so the average number of contagions per each primary infectious case, and they are also able to change the components of the equilibrium points, that is the numbers of each subpopulation at the equilibrium, and the rates the convergence to such equilibrium points. The usual epidemic models are typically based on differential, difference or mixed equations which describe the coupled dynamics of the various subpopulation or, in general, they can include point and distributed delayed dynamics or to be also formulated in a stochastic framework, [7,8,11]. There are also studies for models of networks available which include different nodes which can represent different sets of interacting communities [14,15], which combined control strategies which take into account the communication links and population flows.…”