Gastrointestinal Infarction as a Manifestation of Rheumatoid Vasculitis

Babian, M; Nasef, Samah Ismail; Soloway, Gregory

doi:10.1111/j.1572-0241.1998.119_c.x

Cited by 30 publications

(7 citation statements)

References 7 publications

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“…Gastrointestinal involvement was reported in our study in 7.4% of HCV-related vasculitis patients, which is similar to the rates described in Wegener granulomatosis22 and rheumatoid arthritis-associated vasculitis 23 24. In contrast, GI involvement in HCV-vasculitis patients was lower than in PAN,25 MPA13 and CSS,12 26 in which GI involvement is described in 40–60%, 30–56% and 20–50% of patients, respectively 11.…”

Section: Discussionsupporting

confidence: 86%

Presentation and outcome of gastrointestinal involvement in hepatitis C virus-related systemic vasculitis: a case–control study from a single-centre cohort of 163 patients

Terrier

Saadoun²,

Sène³

et al. 2010

Gut

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Section: Discussionsupporting

confidence: 86%

Presentation and outcome of gastrointestinal involvement in hepatitis C virus-related systemic vasculitis: a case–control study from a single-centre cohort of 163 patients

Terrier

Saadoun²,

Sène³

et al. 2010

Gut

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“…5 patients (50%) showed all 3 of above findings and 5 patients (50%) presented with only one of lished (1-9). The causes for acute bowel ischemia range from embolic, thrombotic or atherosclerotic occlusion of the mesenteric arteries (10,11), primary or secondary thrombotic occlusions of the mesenteric veins (12, 13) certain hematological disorders (12)(13)(14), various forms of vasculitis (14)(15)(16)(17)(18) to various nonocclusive conditions (19)(20)(21).…”

Section: Resultsmentioning

confidence: 99%

“…Therefore, surgical bowel resection is not absolutely necessary (suggesting thromboembolic disease), or just a typical and suggestive clinical setting such as a positive medical history of prior abdominal aortic surgery, retrograde angiography, atrial fibrillation, cardiovascular disease, hypotensive episodes, hematological disorders, vasculitis or known administration of certain drugs (10)(11)(12)(13)(14)(15)(16)(17)(18)(19)(20)(21).…”

mentioning

confidence: 99%

Small bowel ischemia caused by strangulation in complicated small bowel obstruction. ct findings in 20 cases with histo - pathological correlation

Wiesner¹,

Mortelé²

2011

Journal of the Belgian Society of Radiology

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Acute bowel ischemia is a complex entity that may affect both the large and the small bowel and that can result from a variety of different conditions that critically disturb intestinal perfusion (1, 2). The clinical, radiological and histopathological presentation of bowel ischemia may show a wide range. In partial mural bowel ischemia the ischemic damage of the intestine may be limited to the mucosa, or it may be more pronounced and affect also the submucosal space or the muscularis propria. Contrarily, in transmural bowel ischemia (ie. bowel infarction) the ischemic bowel wall damage involves all layers and typically represents a full thickness necrosis of the affected bowel wall.While mild ischemic bowel lesions involving only the mucosa are typically self limiting and reversible, more pronounced ischemic bowel wall damage involving also the submucosal space or the muscularis propria may lead to scarring and strictures as a late complication. Therefore, surgical bowel resection is not absolutely necessary (suggesting thromboembolic disease), or just a typical and suggestive clinical setting such as a positive medical history of prior abdominal aortic surgery, retrograde angiography, atrial fibrillation, cardiovascular disease, hypotensive episodes, hematological disorders, vasculitis or known administration of certain drugs (10-21).However, the majority of histopathologically proven cases of small bowel ischemia in our daily routine represent venous ischemia, caused by extrinsic compression of mesenteric veins in patients with complicated small bowel obstruction, in which the presence of bowel ischemia usually represents only an additional, but surely important finding, since it strongly influences the further management of these patients. It is known, that in context with clinical and radiological findings of acute small bowel obstruction the presence of small bowel wall thickening, ascites and mesenteric stranding at CT has a high sensitivity and specificity for the diagnosis of strangulation induced venous small bowel ischemia (22)(23)(24).However, differentiation between partial mural and transmural bowel ischemia is difficult by CT as long as perforation has not yet occurred and as long as pronounced pneumatosis or portal venous gas do not indicate a more severe and potentially in mild and only partial mural bowel ischemia, whereas transmural bowel infarction definitely requires surgical resection of the necrotic bowel segments.Over the past years CT has become the key imaging modality for the detection of acute bowel ischemia. It is well known that bowel ischemia may present with a wide range of imaging findings at CT including bowel wall thickening, absent or heterogeneous bowel wall enhancement, hypo-or hyperattenuating bowel wall thickening, mesenteric fluid and ascites, pneumatosis or even mesenteric or portal venous gas (2-9). Although many of these CT findings are not highly specific, the diagnosis of intestinal ischemia can be made with a high accuracy by CT if there are additional radiological ...

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“…Individual case reports have described both small bowel and large bowel infarction (Burt et al 1983;Babian et al 1998), appendicitis (Van Laar et al 1998), intrahepatic hemorrhage, intra-abdominal hemorrhage (Achkar et al 1995), and pancreatitis (Geboes and Dalle 2002).…”

Section: Abdominal Vasculitismentioning

confidence: 97%