Genetic disease and amyloid

Hobbs,

doi:10.1136/jcp.s3-12.1.128

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1978

1990

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“…Distinct from connective tissue alterations in growth and maturation, age changes are now believed to be subtle, widespread, and multifactorial. In some circumstances altered immunological reactivity and age may both contribute to the accumulation of the AA protein ofamyloidosis, an accumulation that reminds us ofthe genetic background ofsome rare forms ofthis curious disorder (see J. R. Hobbs (Hobbs, 1978) at page 128). However, to attribute to the molecular anomalies of ageing any particular connective tissue disease such as osteoarthrosis or intervertebral disc disorder (Ball, 1978) remains as much beyond our present state of understanding as does the explanation of most cases of amyloidosis.…”

Section: Summary Of New Knowledgementioning

confidence: 99%