Gold standard approach for removing sialoliths from the submandibular gland

Abstract

Objective Sialolithiasis, the most common disease of the salivary gland, is characterized by calculi in gland and its duct. While most salivary calculi are small in size, giant calculi with diameters spanning several centimeters are also reported occasionally. The surgical removal of recurrent submandibular sialoliths. Results This study presents of recurrent submandibular sialadenitis/sialadenosis and the characteristic signs and symptoms included pain, recurrent swelling, and discharge of pus, fibrotic and atrophic condition of the gland followed by cessation of secretary functions. Radiological examinations revealed large and irregular structures in the reported cases. Conclusion We have described the characteristic clinical and radiological features, diagnosis and treatment approach for submandibular salivary gland sialadenitis/sialadenosis. Keywords chronic sialadenitis/sialadenosis, CT scan, excision of gland, sialoliths of the submandibular salivary gland a b 58

Oxidative‐antioxidant imbalance in chronic sialadenitis of submandibular gland in human and rat

“…Chronic sialadenitis (CS) is a nonneoplastic disease that causes salivary dysfunction, mostly occurring in the submandibular gland (SMG) and parotid gland (Abdulkareem et al, 2018; Delli et al, 2014). Clinically, CS is mostly caused by salivary flow obstruction owing to sialolithiasis, gland inflammation, duct stenosis, external duct compression, mucus plugs or thick saliva sludge (Delagnes et al, 2017).…”

“…Chronic sialadenitis (CS) is a nonneoplastic disease that causes salivary dysfunction, mostly occurring in the submandibular gland (SMG) and parotid gland (Abdulkareem et al, 2018;Delli et al, 2014).…”

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Chronic sialadenitis (CS) is a nonneoplastic disease that causes salivary dysfunction, mostly occurring in the submandibular gland (SMG) and parotid gland (Abdulkareem et al, 2018;Delli et al, 2014).Clinically, CS is mostly caused by salivary flow obstruction owing to sialolithiasis, gland inflammation, duct stenosis, external duct compression, mucus plugs or thick saliva sludge (Delagnes et al, 2017). The histopathological features of CS include acinar atrophy, ductal dilatation, ductal epithelial proliferation and metaplasia, lymphoplasmacytic infiltration, progressive fibrosis and intraductal mucous deposit (Hong et al, 2017).

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