Gradually Progressive Dementia Without Discrete Cerebrovascular Events in a Patient With Sneddon's Syndrome

Wright, R. Alan; Kokmen, Emre

doi:10.4065/74.1.57

Cited by 28 publications

(19 citation statements)

References 19 publications

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“…Further, valid clinical diagnosis and rationally based treatment are in large measure a function of the validity and reliability of medical constructs and their operationalization and classification [5][6][7][8]. Results of our investigations of cognitive impairment in a broad spectrum of vascular disorders indicate that cerebral infarction is not the only path to the phenotypic presentation of vascular dementia [1][2][3][4][9][10][11]. Data are presented that indicate vascular disease without cerebral infarction results in a continuum of cognitive impairment, with one end of this continuum represented by the concept of noninfarct vascular dementia.…”

Section: Introductionmentioning

confidence: 99%

Noninfarct vascular dementia and Alzheimer dementia spectrum

Emery

Gillie

Smith

2005

Journal of the Neurological Sciences

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Section: Introductionmentioning

confidence: 99%

Noninfarct vascular dementia and Alzheimer dementia spectrum

Emery

Gillie

Smith

2005

Journal of the Neurological Sciences

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“…Some, but not all, cases have had associated anticardiolipin antibodies. Vascular dementia has been reported in this syndrome [45].…”

Section: Miscellaneous Other Vascular Cognitive Syndromesmentioning

confidence: 71%

Vascular dementia: A review of recent evidence for prevention and treatment

Kirshner

2009

Curr Neurol Neurosci Rep

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Vascular dementia is a common cause of late-life cognitive decline. Vascular risk factors, transient ischemic attacks, silent and clinically evident strokes, and ischemic changes on brain imaging studies are all associated with the development of dementia. Prevention of vascular dementia involves treatment of risk factors and the same preventive measures used to avoid heart attacks and strokes. Other treatments for vascular dementia, although promising, remain unproved.

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“…İleri dönemlerde orta derecede kognitif bozulma (konsantrasyon bozukluğu gibi) veya ciddi entelektüel bozulma (demans) ya da emosyonel bozukluklar (depresyon, emosyonel labilite gibi) gelişebilir (8,16,17). Kognitif etkilenmenin yaygın iskemiye bağlı vasküler demanstan kaynaklandığı düşünülmektedir (10).…”

Section: Discussionunclassified

Otoantikorları Negatif Sneddon Sendromu: Olgu Sunumu ve Literatürün Gözden Geçirilmesi

Arslan¹,

Dırıcan²,

Ertem³

et al. 2013

npa

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Guillain Barré syndrome (GBS) is a disorder characterized by areflexia and progressive ascending lower extremity weakness. Bilateral peripheral facial diplegia and paresthesia is a rare GBS variant in which the preceding infection, auto-antibody positivity and treatment models are still unclear. In this rare variant, no case with an etiology of varicella infection has been reported yet. Here, we report a case with facial diplegia and parestesia secondary to varicella infection and discuss clinical, electrophysiological and laboratory findings, and review the literature focusing on the latest reports on this disease.

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Gradually Progressive Dementia Without Discrete Cerebrovascular Events in a Patient With Sneddon's Syndrome

Cited by 28 publications

References 19 publications

Noninfarct vascular dementia and Alzheimer dementia spectrum

Noninfarct vascular dementia and Alzheimer dementia spectrum

Vascular dementia: A review of recent evidence for prevention and treatment

Otoantikorları Negatif Sneddon Sendromu: Olgu Sunumu ve Literatürün Gözden Geçirilmesi

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