2004
DOI: 10.1016/j.ejogrb.2004.01.010
|View full text |Cite
|
Sign up to set email alerts
|

Granulosa cell-oocyte interactions

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
3
1
1

Citation Types

0
51
0
4

Year Published

2005
2005
2023
2023

Publication Types

Select...
10

Relationship

1
9

Authors

Journals

citations
Cited by 84 publications
(55 citation statements)
references
References 41 publications
0
51
0
4
Order By: Relevance
“…Oocyte-follicular cell interactions are complicated events in vertebrate folliculogenesis and oogenesis, and ultimately lead to the acquisition of their respective competences through their mutual interactions (Cecconi et al, 2004;Gilchrist et al, 2004;Rolaki et al, 2005). De La Fuente and Eppig (2001) have demonstrated that mouse granulosa cells allow the transfer of about 85% of the oocyte metabolic needs and modulate oocyte transcriptional activity.…”
Section: Discussionmentioning
confidence: 99%
“…Oocyte-follicular cell interactions are complicated events in vertebrate folliculogenesis and oogenesis, and ultimately lead to the acquisition of their respective competences through their mutual interactions (Cecconi et al, 2004;Gilchrist et al, 2004;Rolaki et al, 2005). De La Fuente and Eppig (2001) have demonstrated that mouse granulosa cells allow the transfer of about 85% of the oocyte metabolic needs and modulate oocyte transcriptional activity.…”
Section: Discussionmentioning
confidence: 99%
“…Through cyst and follicle formation, germ cells and their surrounding somatic cells establish an intricate system of mutual interactions that ultimately lead to the acquisition of their respective competences (Cecconi et al, 2004). Previous studies have demonstrated that granulosa cells allow the transfer of about 85% of the oocyte metabolic needs and modulate oocyte transcriptional activity (De La Fuente and Eppig, 2001).…”
Section: Differential Expression and Developmental Behaviour Of Cagcnmentioning
confidence: 99%
“…Among the several regulative pathways involved in the regulation of oogenesis (Edson et al, 2009;Cecconi et al, 1996;Cecconi et al, 2004;Canipari et al, 2012), the PI3K signalling pathway is a critical regulator of follicle growth, differentiation and survival. The observation that deletions of these genes cause infertility and premature ovarian failure (POF) sustains their essential role in preserving the normal female reproductive lifespan (Edson et al, 2009).…”
Section: The Pi3k/akt Gene Family In the Mammalian Ovarymentioning
confidence: 99%