Guillain-barré syndrome following danazol and corticosteroid therapy for hereditary angioedema

Hory, B; Blanc, Dominique; Boillot, A.; Panouse-Perrin, J

doi:10.1016/0002-9343(85)90553-4

Cited by 19 publications

(3 citation statements)

References 27 publications

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“…In our kindreds we have been unable to confirm associations to neurological, renal or autoimmune diseases which have been reported by others [15][16][17][18][19][20][21].…”

Section: Clinical Manifestations Morbidity and Mortalitycontrasting

confidence: 83%

Type I hereditary angio‐oedema. Variability of clinical presentation and course within two large kindreds

Winnewisser

Rossi

Späth

et al. 1997

Journal of Internal Medicine

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Abstract. Winnewisser J, Rossi M, Spa$ th P, Bu$ rgi H (Bu$ rgerspital Solothurn and Bluttransfusionsdienst, Bern, Switzerland). Type I hereditary angio-oedema. Variability of clinical presentation and course within two large kindreds. J Intern Med 1997 ; 241 : 39-46. Objectives.Hereditary angiooedema (C1-inhibitor deficiency) is a disease with protean manifestations which is often misdiagnosed initially. The purpose of this study was to delineate the clinical spectrum and course of this potentially life-threatening disease. Setting. Ten members of two unrelated affected kindreds were repeatedly hospitalized in our service with attacks of angiooedema. Among the 271 members of the two kindreds 69 were identified who reported typical attacks. Design and subjects. Detailed information on the clinical course and laboratory data (C1-inhibitor concentration and activity, C4 concentration) were obtained from 59 of the 69 affected subjects. Results. C1-inhibitor concentration and activity were low in all affected patients. The disease had become manifest by age 30 years in 98 %. Most patients suffered from attacks of subcutaneous swellings and abdominal pain, and half of the patients have had

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“…In our kindreds we have been unable to confirm associations to neurological, renal or autoimmune diseases which have been reported by others [15][16][17][18][19][20][21].…”

Section: Clinical Manifestations Morbidity and Mortalitycontrasting

confidence: 83%

Type I hereditary angio‐oedema. Variability of clinical presentation and course within two large kindreds

Winnewisser

Rossi

Späth

et al. 1997

Journal of Internal Medicine

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“…However, we did investigate drug intake since there are anecdotal reports of GBS onset after drug exposure [3,6,12,18], and the possible etiological role of gangliosides has recently been debated [4,8,16,25]. Drug use was assessed both by questionnaire and by the Regional Drug Register.…”

Section: Discussionmentioning

confidence: 99%

“…Factors reported to trigger GBS are: surgery [1,28], cancer [19], pregnancy [24], autoimmune diseases [22], and drug exposure: captopril [3], danazol [12], zimeldine [6], amitriptyline [18], and gangliosides [4,16,25]. Furthermore, infections from cytomegalovirus [14,27] and Campylobacter jejuni [11,26,29] have been reported preceding GBS.…”

Section: Introductionmentioning

confidence: 99%

Risk factors for Guillain-Barré syndrome: a population-based case-control study

D’Alessandro

Casmiro

Guarino

1999

Journal of Neurology

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We performed the first population-based case-control study on clinical risk factors and drug exposure in Guillain-Barré syndrome (GBS). Sixty patients with GBS were collected through an incidence survey performed in the Emilia-Romagna region of northern Italy. GBS patients were compared with 109 hospital controls (HC) and 59 population controls (PC) as for clinical events and drug exposure during the month preceding the study inclusion. Comparison of HC with PC showed no significant difference in the frequency of clinical events which occurred during the preceding month. This indicated that our HC were well representative of the general population for the risk factors investigated. GBS patients were compared with HC or both HC and PC using both univariate and multivariate analysis. Univariate analysis showed a significant association with fever and upper airway infection symptoms occurring in the preceding month. Furthermore, a significantly higher exposure to antibiotics, antipyretics/analgesics, and gangliosides was observed. However, multivariate analysis showed that only fever was significantly related to GBS onset (OR 28, 95% CI 5.2-313 for GBS vs. HC; OR 29, 95% CI 7.0-256 for GBS vs. HC and PC).

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Drug-Induced Guillain-Barré Syndrome

Jain

2021

Drug-Induced Neurological Disorders

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Guillain-barré syndrome following danazol and corticosteroid therapy for hereditary angioedema

Cited by 19 publications

References 27 publications

Type I hereditary angio‐oedema. Variability of clinical presentation and course within two large kindreds

Type I hereditary angio‐oedema. Variability of clinical presentation and course within two large kindreds

Risk factors for Guillain-Barré syndrome: a population-based case-control study

Drug-Induced Guillain-Barré Syndrome

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