Heat-related deaths

Byard, Roger W.

doi:10.1007/s12024-013-9413-y

Cited by 5 publications

(2 citation statements)

References 19 publications

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“…The onset and development of both 'skin slippage' and 'marbling' are considered to be nonspecific to temperature conditions and have previously been found to occur in both extreme cold and heat-related deaths [23,43]. It is therefore unlikely that mortuary refrigeration inhibited the development of these markers.…”

Section: Scene and Autopsy Decompositionmentioning

confidence: 99%

Decomposition variability between the scene and autopsy examination and implications for post-mortem interval estimations

Giles

Errickson

Márquez‐Grant

2022

Journal of Forensic and Legal Medicine

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Section: Scene and Autopsy Decompositionmentioning

confidence: 99%

Decomposition variability between the scene and autopsy examination and implications for post-mortem interval estimations

Giles

Errickson

Márquez‐Grant

2022

Journal of Forensic and Legal Medicine

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“…If the subject dies more slowly, macroscopic findings include pulmonary and cerebral oedema associated with diffuse neuronal damage, subarachnoid and subdural haemorrhages and petechial haemorrhages, mostly in periventricular areas. 16 Moreover, polyvisceral congestion, especially involving the lungs and brain, can be found. On histology, brain involvement is represented by focal degenerative lesions in the cerebral cortex, basal ganglia and brain stem.…”

Section: Introductionmentioning

confidence: 99%

Death following extreme temperature exposure: Histological, biochemical and immunohistochemical markers

et al. 2021

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Introduction Defining extreme temperatures as the cause of death remains challenging. It is mostly based on circumstantial, macroscopic and microscopic features. Methods We retrospectively compared groups of cases of fatal hypothermia, fatal hyperthermia and non-extreme temperature-related deaths. We analysed specific histological findings, focusing on samples from the liver, pancreas and kidney. Results Between 1 January 2013 and 31 December 2016, 15 autopsies were performed for deaths related to extreme temperatures. They included 11 cases of fatal hypothermia (group A), four cases of fatal hyperthermia (group B) and eight controls (group C). Perinuclear hepatocyte vacuolisation was observed in seven cases of hypothermia, one case of hyperthermia and four controls. Pancreatic cytoarchitecture was well preserved in two cases of hypothermia, one case of hyperthermia and two controls. No particular microscopic feature was found in pancreatic samples. Renal epithelial tubular cell vacuolisation was observed in seven cases of hypothermia and one case of hyperthermia, while it was absent in all controls. Chromogranin A (CgA) was markedly positive in the pancreatic tissue of five cases of fatal hypothermia and one control, and mildly positive in one case of fatal hyperthermia. No significant p-values were observed for any comparisons ( p > 0.05), except when hypothermia cases group were compared to the control group for the Armanni–Ebstein phenomenon test ( p = 0.0078). Conclusions Although our study did not find a specific microscopic marker, hepatocyte vacuolisation, the Armanni–Ebstein phenomenon and pancreatic CgA positivity, taken together, may be useful tools to confirm hypo- and hyperthermia-related deaths, in addition to circumstantial and macroscopic findings.

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Children: Noninflicted Causes of Childhood Death

Byard

2016

Encyclopedia of Forensic and Legal Medicine

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Heat-related deaths

Cited by 5 publications

References 19 publications

Decomposition variability between the scene and autopsy examination and implications for post-mortem interval estimations

Decomposition variability between the scene and autopsy examination and implications for post-mortem interval estimations

Death following extreme temperature exposure: Histological, biochemical and immunohistochemical markers

Children: Noninflicted Causes of Childhood Death

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