Hipertensión pulmonar e hipertiroidismo en una mujer gestante

Romero, Francisco Botella; Peláez, Juan Manuel Fernández; Díez, A. Gato; López, Almudena López

doi:10.1016/s0025-7753(01)72084-2

Cited by 2 publications

(4 citation statements)

References 7 publications

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“…Table 1 summarizes data on our patients and other reported cases [12, 13, 14, 15, 16, 17, 18, 19, 20, 21, 22]. Included are 4 patients who demonstrated clinical improvement with treatment of hyperthyroidism, but who were without repeat pulmonary artery measurements [20, 21, 22].…”

Section: Discussionmentioning

confidence: 99%

“…Treatment to reduce the effects of excess thyroid hormone, either surgically or medically, results in improvement in the systolic PAP. Improvement in pulmonary pressures occurs after as little as 1 month of therapy [12, 18]. Most, but not all experience reduction in systolic PAP to below 30 mm Hg (table 1).…”

Section: Discussionmentioning

confidence: 99%

“…Females have comprised all previously reported cases except for one [12, 13, 14, 15, 16, 17, 18, 19]. We have encountered 3 male patients with hyperthyroidism and thyrotoxicosis, and the following is a report of these cases.…”

Section: Introductionmentioning

confidence: 95%

“…However, pulmonary hypertension is an acknowledged, albeit often un-investigated effect of thyrotoxicosis or hyperthyroidism [6]. The association between these two conditions has become more evident with increasing reports of improvement in pulmonary hypertension with treatment of hyperthyroidism [12, 13, 14, 15, 16, 17, 18, 19]. …”

Section: Introductionmentioning

confidence: 99%

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Pulmonary Hypertension in Men with Thyrotoxicosis

Soroush-Yari

Burstein²,

Hoo³

et al. 2005

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Thyrotoxicosis has a myriad of respiratory symptoms including dyspnea. Pulmonary hypertension may contribute to the respiratory symptoms of thyrotoxicosis, but is often unrecognized. We describe 3 male patients with thyrotoxicosis and associated pulmonary hypertension. Case reports of an additional 15 patients are also reviewed. In patients with thyrotoxicosis and pulmonary hypertension, treatment of thyrotoxicosis alone is associated with improvement in pulmonary hypertension. Previous reports have consisted of mostly female patients, but we report 3 men. When all cases are considered, the typical patient is female (10/14 = 71%), middle-aged (48 years), with mean pulmonary artery systolic pressures improving from 56 to 32 mm Hg with treatment. Autoantibodies were detected in 10/14 (71%) patients. The response to treatment (medical or surgical) of thyrotoxicosis supports the hypothesis that hyperthyroidism is either a cause of pulmonary hypertension, or a factor that may unmask pulmonary hypertension. Recognition is important since treatment and response are very different compared to other patients with pulmonary hypertension. This association may not be readily considered in men, since most reports have been of women.

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