Histamine Catabolism during Treatment with Chlorpromazine

Johansson, M.-B.; Lindell, S.‐E.; Roos, B. E.; Westling, H.

doi:10.1159/000135020

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Autonomic Drugs1

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1963

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“…Little, if any, metabolism of hexamethonium, or its sulfonium or monoquaternary analogues occurs in the mouse (114). Inoue (88) has provided suggestive evidence for the formation in vivo of the N-glu curonides of histamine by man and rabbits, and Johansson et al (91) have indicated that large doses of chlorpromazine have no effect on the pattern of urinary excretion of histamine and its metabolites by man. Mayer (121) has investigated the fate of dichloroisoproterenol ({3-hydroxy· N -isopropyl-3,4-dichlorophenethylamine), labeled in the {3-position with 3H, in dogs and mice.…”

Section: Autonomic Drugsmentioning

confidence: 99%

Metabolic Fate

Shideman¹,

Mannering²

1963

Annu. Rev. Pharmacol.

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1 The survey of the literature pertaining to this review was concluded in May 1962.2 Abbreviations used in this chapter include: ADH (alcohol dehydrogenase); AT (3-amino-l,2,4-triazole); CoA (coenzyme A); NAD (nicotinamide-adenine dinucleotide); NADH2 (nicotinamide-adenine dinucleotide reduced form); NADP (nicotinamide-adenine dinucleotide phosphate); NADPH2 (nicotinamide-adenine dinucleotide phosphate reduced form); TEPA (triethylenephosphoramide).

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Section: Autonomic Drugsmentioning

confidence: 99%