Histamine‐N‐Methyl Transferase Polymorphism and Risk for Migraine

García‐Martín, Elena; Martı́nez, Carmen; Serrador, Mercedes; Alonso‐Navarro, Hortensia; Navacerrada, Francisco; Agúndez, José A. G.; Jiménez‐Jiménez, Félix Javier

doi:10.1111/j.1526-4610.2007.01056.x

Cited by 25 publications

(18 citation statements)

References 28 publications

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“…Recruitment of patients was done in the general neurological clinics of 3 University Hospitals during the period between September 2006 and August 2007 (the onset of migraine attacks was under age 15 years in 113 patients, positive family history for migraine was reported by 147 patients, and 98 of them had MWA). Most of these patients have participated previously in other genetic association studies reported by our group …”

Section: Methodsmentioning

confidence: 99%

Gamma‐Aminobutyric Acid (Gaba) Receptors Rho (Gabrr) Gene Polymorphisms and Risk for Migraine

et al. 2017

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Section: Methodsmentioning

confidence: 99%

Gamma‐Aminobutyric Acid (Gaba) Receptors Rho (Gabrr) Gene Polymorphisms and Risk for Migraine

et al. 2017

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“…All eligible patients were invited to participate and all of them agreed to do so. These patients participated in other genetic association studies by our group …”

Section: Methodsmentioning

confidence: 99%

Diamine Oxidase rs10156191 and rs2052129 Variants Are Associated With the Risk for Migraine

et al. 2015

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“…In the realm of genetics and migraine, it was hypothesized that an association between migraine and polymorphisms of the gene encoding histamine N‐methyltransferase, the enzyme which degrades histamine in the CNS, might exist. However, a recent study has failed to demonstrate this 208 …”

Section: Histamine and Headachementioning

confidence: 99%

Chemical Mediators of Migraine: Preclinical and Clinical Observations

2011

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Migraine is a neurovascular disorder, and although the pathophysiology of migraine has not been fully delineated, much has been learned in the past 50 years. This knowledge has been accompanied by significant advancements in the way migraine is viewed as a disease process and in the development therapeutic options. In this review, we will focus on 4 mediators (nitric oxide, histamine, serotonin, and calcitonin gene-related peptide) which have significantly advanced our understanding of migraine as a disease entity. For each mediator we begin by reviewing the preclinical data linking it to migraine pathophysiology, first focusing on the vascular mechanisms, then the neuronal mechanisms. The preclinical data are then followed by a review of the clinical data which support each mediator’s role in migraine and highlights the pharmacological agents which target these mediators for migraine therapy.

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Histamine‐N‐Methyl Transferase Polymorphism and Risk for Migraine

Abstract: The results of the present study suggest that HNMT polymorphism in not related with the risk for migraine.

Cited by 25 publications

References 28 publications

Gamma‐Aminobutyric Acid (Gaba) Receptors Rho (Gabrr) Gene Polymorphisms and Risk for Migraine

Gamma‐Aminobutyric Acid (Gaba) Receptors Rho (Gabrr) Gene Polymorphisms and Risk for Migraine

Diamine Oxidase rs10156191 and rs2052129 Variants Are Associated With the Risk for Migraine

Chemical Mediators of Migraine: Preclinical and Clinical Observations

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