Histological evaluation of a tooth with hyperplastic pulpitis and periapical osteosclerosis

Abstract

Summary Histological examination was carried out in a case of hyperplastic pulpitis associated with periapical osteosclerosis.

Histological evaluation of teeth with hyperplastic pulpitis caused by trauma or caries: case reports

“…Çalışkan et al . (1997) demonstrated that radicular pulp tissue in cases of chronic hyperplastic pulpitis with periapical osteosclerosis also showed fibrosis with absence of inflammatory cells.…”

“…Presumably the young pulp does not become necrotic following exposure, because its natural defences and rich supply of blood allow it to resist bacterial infection (Kim & Trowbridge 1987). This reaction is probably favoured by free exposure of the pulp in complicated crown fracture or in teeth whose crowns are completely destroyed by caries, permiting continuous salivary rinsing and preventing impaction of contaminated debris (Cvek et al . 1982, Çalışkan et al .…”

“…Therefore, an active dressing was considered necessary for healing. However, the epithelial layer over the surface of the polyp protects the underlying granulation tissue from the harmful effects that will disturb wound healing in the oral cavity (Çalışkan et al . 1997).…”

“…The tissue in the pulp chamber is often transformed into granulation tissue, which projects from the pulp into the carious lesion. There may be fibrosis and calcific degeneration in some areas of the coronal pulp, whilst the radicular pulp tissue may be healthy or contain few chronic inflammatory cells (Walton et al . 1985, Grossman et al .…”

Exposed human pulp caused by trauma and/or caries in primary dentition: a histological evaluation

“…The results of our study show only two teeth in which polyps were covered with epithelium. Ç als¸kan et al (27) have suggested that the epithelial layer over the surface of the polyp protects the underlying granulation tissue from the harmful effects that will disturb wound healing in the oral cavity. These defensive reactions probably contribute to the inherent healing potential of young dental pulp in which hyperplastic pulpitis develops (5).…”