Hydrocephalus related to pulsion diverticulum of lateral ventricle.

Mott., May Alzada; Cummins, Brian H.

doi:10.1136/adc.49.5.407

Cited by 19 publications

(7 citation statements)

References 3 publications

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“…Ventricular rupture can occur in many sites. The most commonly described rupture occurs along the medial wall of the atrium of the lateral ventricle, which is supported by the splenium of the corpus callosum above and behind, the crura of the fornices and the alvei and fimbriae of the hippocampus . Due to increasing intraventricular pressure, there is destruction of the ependymal lining but preservation of the pial interface, leading to the development of pia lined diverticula…”

Section: Discussionmentioning

confidence: 99%

Congenital aqueduct stenosis: Progressive brain findings in utero to birth in the presence of severe hydrocephalus

et al. 2018

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Hydrocephalus in the fetus results in enlarging ventricular rupture, loss of the septum pellucidum leaflets, volume reduction of brain parenchyma including corpus callosum, and risk for Chiari I anomaly. Given advances in fetal surgery and imaging in the last 3 decades, there may be cause to revisit the idea of in utero cerebral spinal fluid diversion as a means to potentially ameliorate progressive loss of the developing brain.

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Section: Discussionmentioning

confidence: 99%

Congenital aqueduct stenosis: Progressive brain findings in utero to birth in the presence of severe hydrocephalus

et al. 2018

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“…Most of these theories have had to be revised since the identification of the anatomical structure responsible for upward gaze, which is located in the periaqueductal gray matter ventral to the aqueduct, in the dorsal interstitial nucleus of the medial longitudinal fasciculus. 7,48 The region of the upper brainstem as well as all structures located in or around the notch of the tentorium are known to be subject to significant anatomical modifications due to pressure variations across the tentorium; 4,9,18,23,[29][30][31][32][33][37][38][39]42,43,49,58,60,62 this is typical in obstructive hydrocephalus due to aqueductal stenosis. All these changes can be explained by the existence of a long-standing pressure gradient across the tentorium, with higher pressure levels in the supratentorial compartment and lower pressure levels in the posterior fossa.…”

Section: Discussionmentioning

confidence: 99%

Sylvian aqueduct syndrome and global rostral midbrain dysfunction associated with shunt malfunction

Cinalli

Sainte‐Rose²,

Simon³

et al. 1999

Journal of Neurosurgery

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It is probable that in obstructive hydrocephalus, at the time of shunt malfunction, the development of a transtentorial pressure gradient could initially induce a functional impairment of the upper midbrain, inducing upward gaze palsy. The persistence of the gradient could lead to a global dysfunction of the upper midbrain. Third ventriculostomy contributes to equalization of cerebrospinal fluid pressure across the tentorium by restoring free communication between the infratentorial and supratentorial compartments, resulting in resolution of the patient's clinical symptoms.

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“…1, 4,6,8,14,18,19,[21][22][23]26,28,29,31 Nevertheless, clinically overt cerebellar ataxia develops only in r a r e o c c a s i o n s . 4'8A4'19'23'26'28 Furthermore, the disappearance of this symptom after surgery has been documented in only one previous case.…”

Section: Discussionmentioning

confidence: 99%