2014
DOI: 10.4103/0972-5229.125439
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Hyperkalemia: A rare cause of acute flaccid quadriparesis

Abstract: Acute flaccid quadriparesis secondary to hyperkalemia is a very rare and serious but reversible medical emergency. We present a case of a 73-year-old female who was admitted with rapidly progressive ascending paraparesis progressing to quadriparesis in about 10 h due to hyperkalemia. Patient was treated with antihyperkalemic measures. Her power improved dramatically as potassium levels normalized and she had an uneventful recovery.

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Cited by 8 publications
(5 citation statements)
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“…The major symptoms being osteotendinous hyporeflexia, quadriparesis/ paralysis, more or less associated with respiratory damage and sensory loss. Complete recovery of the neurological deficit occurred after correction of hyperkalaemia by medicinal means, whether or not associated with extra-renal purification [7][8][9][10].…”
Section: Discussionmentioning
confidence: 99%
“…The major symptoms being osteotendinous hyporeflexia, quadriparesis/ paralysis, more or less associated with respiratory damage and sensory loss. Complete recovery of the neurological deficit occurred after correction of hyperkalaemia by medicinal means, whether or not associated with extra-renal purification [7][8][9][10].…”
Section: Discussionmentioning
confidence: 99%
“…Hyperkalemia, defined as a serum potassium level greater than 6 mmol/L, is a potentially life-threatening electrolyte abnormality seen in patients secondary to either dietary intake or impaired excretion of potassium [ 3 ]. Patients with severe hyperkalemia can have life-threatening symptoms such as cardiac dysrhythmias and conduction abnormalities, as well as muscle weakness that can progress to paralysis [ 4 ]. The paralysis associated with hyperkalemia starts distally in the lower extremities and ascends to involve the patient’s trunk and upper extremities with little to no sensory impairment [ 5 ].…”
Section: Discussionmentioning
confidence: 99%
“…Neste contexto, a RTA distal (clássica ou tipo 1) atua nos néfrons distais e não permitem que estes exerçam a função de secreção dos íons H+, por isso diminui o pH urinário, gerando a acidose metabólica (Singh et al, 2014-. Varshney et al, 2015.…”
Section: Discussionunclassified
“…bicarbonato pelo túbulo distal (RTA tipo 1), ou por falha em reabsorver bicarbonato no túbulo proximal (RTA tipo 2) ou incapacidade de excretar potássio no ducto coletor (RTA tipo 4) (Palmer et al, 2020-Nicoletta & Schwartz, 2004.Clinicamente, a RTA tipo 1, pode se apresentar com fraqueza muscular severa de membros inferiores e superiores, distúrbio ácido-básico, a exemplo da acidose metabólica e hipocalemia grave (Singh et al, 2014).…”
unclassified