1950
DOI: 10.1016/0030-4220(50)90192-7
|View full text |Cite
|
Sign up to set email alerts
|

Hypoplasia of the mandible (Pierre Robin syndrome) with complete cleft palate

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
1

Citation Types

0
1
0

Year Published

1960
1960
2024
2024

Publication Types

Select...
6

Relationship

0
6

Authors

Journals

citations
Cited by 8 publications
(1 citation statement)
references
References 5 publications
0
1
0
Order By: Relevance
“…[ 10 ] PRS, in association with other genetic syndromes, may carry a very high recurrence risk. In the case of PR anomalad, the primary defect lies in the arrested development and ensuing hypoplasia of the mandible, ultimately producing the characteristic “bird facies.”[ 11 ] This in turn prevents the normal descent of the tongue between the palatal shelves, resulting in a cleft palate. [ 12 ] Airway obstruction and feeding difficulty associated with PRS require invasive therapeutic measures.…”
Section: Discussionmentioning
confidence: 99%
“…[ 10 ] PRS, in association with other genetic syndromes, may carry a very high recurrence risk. In the case of PR anomalad, the primary defect lies in the arrested development and ensuing hypoplasia of the mandible, ultimately producing the characteristic “bird facies.”[ 11 ] This in turn prevents the normal descent of the tongue between the palatal shelves, resulting in a cleft palate. [ 12 ] Airway obstruction and feeding difficulty associated with PRS require invasive therapeutic measures.…”
Section: Discussionmentioning
confidence: 99%