2022
DOI: 10.2139/ssrn.4186473
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Hypoxia/Inflammation-Induced Upregulation of HIF-1α and C/EBPβ, Promoted Nephroblastoma EMT by Improving HOXA11-AS Transcription

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(1 citation statement)
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“…In the regulation of inflammation, HOXA11-AS mediated CaOx crystal-induced renal inflammation via the miR-124-3p/MCP-1 axis [41], while inhibition of HOXA11-AS repressed neuroinflammation and neuronal apoptosis in Parkinson's disease model through through miR-124-3p-FSTL1-NF-κB axis [42] and miR-98-5p/ EphA4 [43]. In addition, induction of HOXA11-AS by hypoxia/inflammation upregulated of HIF-1α and C/ EBPβ further to promote epithelial-mesenchymal transition ability of nephroblastoma [44]. All these evidences indicate that HOXA-11 acts as a proinflammatory factor.…”
Section: Discussionmentioning
confidence: 99%
“…In the regulation of inflammation, HOXA11-AS mediated CaOx crystal-induced renal inflammation via the miR-124-3p/MCP-1 axis [41], while inhibition of HOXA11-AS repressed neuroinflammation and neuronal apoptosis in Parkinson's disease model through through miR-124-3p-FSTL1-NF-κB axis [42] and miR-98-5p/ EphA4 [43]. In addition, induction of HOXA11-AS by hypoxia/inflammation upregulated of HIF-1α and C/ EBPβ further to promote epithelial-mesenchymal transition ability of nephroblastoma [44]. All these evidences indicate that HOXA-11 acts as a proinflammatory factor.…”
Section: Discussionmentioning
confidence: 99%